Thanks for the heads up on sterility. If I proceed with this idea, I'll make sure to run the compound through a syringe filter to make sure there are no contaminants. I'm already taking testosterone replacement at 70 mg a week, which is on the low side for TRT, so the 0.5 mg dosages Patient 1 was taking with each injection will make almost no difference in my hormone profile. The whole idea is to localize the androgenic effect in the penis. My PGE should arrive in a few days. I'll keep investigating for a while before taking the plunge.kingsnake;534083 said:
The snake man's chemical PE log
- Thread starter kingsnake
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88mans PGE-1 Chemical Enlargement and Jelq Research Project
My PGE-1 log^. If you want to follow it would be much appreciated. Thanks.
My PGE-1 log^. If you want to follow it would be much appreciated. Thanks.
Hey therulersback and bigdex28. I make sure to wash off good after using the dht gel and dmso to prevent any contamination to anyone else.
Hey agent7x6. Good luck when you pge1 arrives. Let me know if you need any help with using it and what to expect with your erection workouts.
Hey 88man. Good thread you got there bro. I’ll definitely be keeping tabs on your progress.
Hey fellas so two days ago I upped the pge1 dosage to an all time high for me being 50 mcgs. The pain was utterly amazing. Extreme torture 10 minutes after injecting. To make matters worse I kept my silicone sleeve and cable clamp on as a cockring for 1 hour to keep my penis saturated in pge1 and to keep the rest of the chems (igf-des, tb-500) local in the shaft for as long as possible.
I didn’t have the clamp on tight like a real clamping workout. I just had it on tight enough to keep the chems trapped but man did my dick swell like no other. During the first hour of my pge1 erection, I would take off the clamp for 5 minutes at a time every 10 to 15 minutes to get circulation back then reapply it. The pain was excruciating.
I had to run some bath water and sit in the tub to let my dick sooth from the immense pain of 50 mcgs of pge1.
After 1 hour went by, I couldn’t take it any more and removed the silicone sleeve and the clamp. After removing the clamp and the sleeve my erection went down to about 60 to 70% lasted for another hour for a total of 2 hours of pge1 erection time. Not bad. Im still not satisfied though. Im looking for 2 to 3 hours or even 4 hours of erection time so next time I’ll increase the dosage to 55 mcgs.
Also, Im in the process of ordering some phentolamine and forskolin. Phentolamine is a vasodilator used in different erection medications. It grants a nice strong painless erection all by itself which is great for having sex with. However it doesn’t have any tunica delinking properties like pge1. I will be stacking phentolamine with pge1 in my injection cocktail. PGE1 erections right now bring me to 60 to 80% erection level if Im not physically stimulating myself. With the use of phentolamine working in synergy with the pge1, the erection strength will most definitely increase up to 90 to 95% without any needed physical or sexual stimulation.
A 90% erection maintained for at least 1 to 2 hours will cause more internal pressure and expansion to the tunica as its collagen bonds are delinked by the pge1. Im looking for 2 hours of being 90 to 95% erect then 1 to 2 hours of being 70 to 50% erect as the erection dies down. So the phentolamine’s main purpose for me will be to increase my erection strength working with pge1 for a better internal stretch workout while the pge1 delinks collagen.
The forskolin that I will be adding is to increase my sensitivity to pge1 so my need to increase in pge1 will slow down a bit. I still want to reach over 100mcgs of pge1 for 2 to 4 hour erections since the more pge1 in the penis, the more collagen delinking happens. I just don’t want to reach such high levels so fast. I need time to let my pain tolerance catch up to PGE1 aka liquid PAIN.
Boy I remember back in the day when just 15 mcgs injected any time of the day would grant me a nice painful 3 to 4 hour erection. Now with more size in girth and more created receptor sites as a result, my pge1 dosage continues to increase to meet the demand.
Hey agent7x6. Good luck when you pge1 arrives. Let me know if you need any help with using it and what to expect with your erection workouts.
Hey 88man. Good thread you got there bro. I’ll definitely be keeping tabs on your progress.
Hey fellas so two days ago I upped the pge1 dosage to an all time high for me being 50 mcgs. The pain was utterly amazing. Extreme torture 10 minutes after injecting. To make matters worse I kept my silicone sleeve and cable clamp on as a cockring for 1 hour to keep my penis saturated in pge1 and to keep the rest of the chems (igf-des, tb-500) local in the shaft for as long as possible.
I didn’t have the clamp on tight like a real clamping workout. I just had it on tight enough to keep the chems trapped but man did my dick swell like no other. During the first hour of my pge1 erection, I would take off the clamp for 5 minutes at a time every 10 to 15 minutes to get circulation back then reapply it. The pain was excruciating.
I had to run some bath water and sit in the tub to let my dick sooth from the immense pain of 50 mcgs of pge1.
After 1 hour went by, I couldn’t take it any more and removed the silicone sleeve and the clamp. After removing the clamp and the sleeve my erection went down to about 60 to 70% lasted for another hour for a total of 2 hours of pge1 erection time. Not bad. Im still not satisfied though. Im looking for 2 to 3 hours or even 4 hours of erection time so next time I’ll increase the dosage to 55 mcgs.
Also, Im in the process of ordering some phentolamine and forskolin. Phentolamine is a vasodilator used in different erection medications. It grants a nice strong painless erection all by itself which is great for having sex with. However it doesn’t have any tunica delinking properties like pge1. I will be stacking phentolamine with pge1 in my injection cocktail. PGE1 erections right now bring me to 60 to 80% erection level if Im not physically stimulating myself. With the use of phentolamine working in synergy with the pge1, the erection strength will most definitely increase up to 90 to 95% without any needed physical or sexual stimulation.
A 90% erection maintained for at least 1 to 2 hours will cause more internal pressure and expansion to the tunica as its collagen bonds are delinked by the pge1. Im looking for 2 hours of being 90 to 95% erect then 1 to 2 hours of being 70 to 50% erect as the erection dies down. So the phentolamine’s main purpose for me will be to increase my erection strength working with pge1 for a better internal stretch workout while the pge1 delinks collagen.
The forskolin that I will be adding is to increase my sensitivity to pge1 so my need to increase in pge1 will slow down a bit. I still want to reach over 100mcgs of pge1 for 2 to 4 hour erections since the more pge1 in the penis, the more collagen delinking happens. I just don’t want to reach such high levels so fast. I need time to let my pain tolerance catch up to PGE1 aka liquid PAIN.
Boy I remember back in the day when just 15 mcgs injected any time of the day would grant me a nice painful 3 to 4 hour erection. Now with more size in girth and more created receptor sites as a result, my pge1 dosage continues to increase to meet the demand.
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let me suggest something to help with your rapidly increasing tolerance to pge1....which i also am experiencing. I while back i read a couple of studies suggesting that α1-selective alpha blockers can markedly increase the potency of pge-1 induced erections. Doxazosin (a drug used to treat high blood pressure and benign prostatic hyperplasia) in particular was shown to be effective at the 4mg dose.
I'm sorry i dont have the links to the studies. but a search on PubMD might help.
the study said that 4mg oral doxazosin with pge-1 injections produced erections in men in which pge-1 alone failed.
I'm sorry i dont have the links to the studies. but a search on PubMD might help.
the study said that 4mg oral doxazosin with pge-1 injections produced erections in men in which pge-1 alone failed.
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Hey snakes, been following your thread since the beginning and was wanting to say thanks for putting this all out there/here.
Had a cluster of questions I wanted to throw out there if you don't mind. You seem to have better "keyword" search terms you use for searching for items that might suit Penis Enlargement.
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Question 1: Was curious if you have ever looked into helichrysum italicum essential oil as a topical with DMSO? A lot of people report that it has amazing effects on scar tissue reduction. Was researching it for cell/nerve regeneration. Not sure what affect it may have on Penis Enlargement and actually adding to anything regarding gains.
Question 2: Are you using anything special for jelqing(oil/lube wise)?
Somewhat of a Question 3: I was also playing with the idea of jelqing with a primrose oil (16oz for around $18, higher linolenic, but lower GLA than borage by 10-15%(?)) and maybe with a magnesium chloride concoction (mag = vasodilator / angiogenic). Maybe this idea has or hasn't cross your mind, was curious.
Question 4: Have you looked into low molecular hyaluronic acid for adding to a DMSO+PABA topical application?
Then there is frankincense essential oil. It looks to have around the same effects as helichrysum, but I seen that it was antiangiogenic for tumor cells. Not sure if that will correlate to the cells needed for Penis Enlargement purposes (ie: being counter-productive) though.
-----
Thanks again.
Had a cluster of questions I wanted to throw out there if you don't mind. You seem to have better "keyword" search terms you use for searching for items that might suit Penis Enlargement.
-----
Question 1: Was curious if you have ever looked into helichrysum italicum essential oil as a topical with DMSO? A lot of people report that it has amazing effects on scar tissue reduction. Was researching it for cell/nerve regeneration. Not sure what affect it may have on Penis Enlargement and actually adding to anything regarding gains.
Question 2: Are you using anything special for jelqing(oil/lube wise)?
Somewhat of a Question 3: I was also playing with the idea of jelqing with a primrose oil (16oz for around $18, higher linolenic, but lower GLA than borage by 10-15%(?)) and maybe with a magnesium chloride concoction (mag = vasodilator / angiogenic). Maybe this idea has or hasn't cross your mind, was curious.
Question 4: Have you looked into low molecular hyaluronic acid for adding to a DMSO+PABA topical application?
Then there is frankincense essential oil. It looks to have around the same effects as helichrysum, but I seen that it was antiangiogenic for tumor cells. Not sure if that will correlate to the cells needed for Penis Enlargement purposes (ie: being counter-productive) though.
-----
Thanks again.
Someone needs to write a disclaimer that this approach should only be taken if all other forms of Penis Enlargement are properly applied for proper amounts of time without any results.
I couldn't agree more. This more than anything else is where newbies will screw themselves up.
Hey markj71. Thanks for the info bro. I’ll definitely be looking into this.
Hey smerc. Yeah I’ve actually been looking into helichrysum italicum essential oils for a minute now. I’ve been researching to see if it can dissolve with DMSO for a better transdermal carry over through the skin into the tunica.
With jelqing I just use coco butter lotion. Nothing special.
With Magnesium. I’ve also been studying on the injections of this stuff for its angiogenic properties. Coughroniellecough was injecting this into his penis for a brief time but later moved on to other chems to mask the pain of pge1.
I’ve actually just ordered some nattokinase powder and bromelain powder to help with collagen remodeling and to break down any scar tissue.
Nattokinase and bromelain are both enzymes that dissolve scar tissue, plaque, and even eats away blood clots. These two compounds are being used for a lot of different things.
Heres a link talking about Nattokinase, bromelain and serrapeptase.
Its use to treat peryonies and other fibrogenic disorders.
http://peyronies-disease-help.com/p...nol-nattokinase-fibrozym-peyronies-treatment/
What Im trying to find out is what the molecular weight of these compounds are to see if they can be transdermally carried through the skin with DMSO.
Also Im still researching to see if powdered acetyl carnitine and powdered vitamin E can dissolve in DMSO.
Hey 88man. Thanks bro. I actually do have a few questions if you don’t mind. After this Sunday coming up, I have one more week on dht gel.
My plan was to use finasteride for 1 month during my break from dht. Finasteride blocks dht in the body and is use for treating male pattern baldness through blocking dht.
After skyping with coughroniellecough strongly advised me to cycle finasteride to block my dht levels for one month. This will increase androgen sensitivity since its being starved of dht for 4 weeks. Then after 4 weeks, Im to hit the dht gel but with extremely high androgen sensitivity from having my dht blocked gains are suppose to skyrocket rapidly during the dht cycle.
Here’s a link to the original Penis Enlargement patent talking about cycling finasteride with dht with your Penis Enlargement routine.
http://www.chemicalenlargement.com/cpereport/cpe1.pdf
Coughroniellecough cycled DHT and finasteride 3 times and he had to stop because his girth reached 7 inches in thickness being to big for most of his clients to handle.
So here’s my question 88man. After reading about finasteride, Im coming across a lot of horror stories on men using it to restore there hair but are reporting permanent libido loss, erectile dysfunction and depression. After reading up on dht blocking hair restoration drugs like finasteride Im leaning towards not doing it in fear of permanently messing up my hormones.
So what Im looking for a safer way to block dht for 4 weeks instead of taking finasteride aka proscar and other dht blocking drugs. So far the only natural supplement that I’ve read on that blocks dht is saw palmetto.
Do you know anything about this 88man. Thanks in advance bro.
Hey MikeShlort and neognostic. I agree. I definitely don’t want any newbies diving into what Im doing without the proper knowledge and mess them selves up.
Ok fellas. Yesterday my injection cocktail was 30 mcgs of igf-des, 100mcgs of tb-500 and my all time high of 57 mcgs of PGE1. The pain of my erection was excruciating the most painful experience I’ve experienced so far. I managed to keep my cable clamp aka cockring on for 40 minutes before taking it off to ensure full pge1 saturation. While wearing the cockring my erection was swollen with red glans and extreme girth ocompanied with extreme throbbing pain that had me in agony.
After taking off the cockring/cable clamp with silicone sleeve, my erection died down to 60 to 70% for 20 minutes then it died down to 40% which totally sucked because even at 40% erect, I was in painful agony as the pge1 circulated through my penis. I was determined to get 2 hours of erection time so I painfully edged to stay at an 70 to 80% erection level.
Im thinking the extreme pain is what’s causing my penis to not stay over 50% erect after taking off the cockring because there were times that when I tried to mentally block out the pain and relax and my chemical erection would sprout back up to 70% without any physical stimulation.
So this evening my dosage will increase yet again hoping for that 2 to 4 hour erection. This evening’s pge1 dosage will be 64 mcgs of liquid PAIN. Wish me luck later on today fellas. Im going to need it. This shit sucks.
Hey smerc. Yeah I’ve actually been looking into helichrysum italicum essential oils for a minute now. I’ve been researching to see if it can dissolve with DMSO for a better transdermal carry over through the skin into the tunica.
With jelqing I just use coco butter lotion. Nothing special.
With Magnesium. I’ve also been studying on the injections of this stuff for its angiogenic properties. Coughroniellecough was injecting this into his penis for a brief time but later moved on to other chems to mask the pain of pge1.
I’ve actually just ordered some nattokinase powder and bromelain powder to help with collagen remodeling and to break down any scar tissue.
Nattokinase and bromelain are both enzymes that dissolve scar tissue, plaque, and even eats away blood clots. These two compounds are being used for a lot of different things.
Heres a link talking about Nattokinase, bromelain and serrapeptase.
Its use to treat peryonies and other fibrogenic disorders.
http://peyronies-disease-help.com/p...nol-nattokinase-fibrozym-peyronies-treatment/
What Im trying to find out is what the molecular weight of these compounds are to see if they can be transdermally carried through the skin with DMSO.
Also Im still researching to see if powdered acetyl carnitine and powdered vitamin E can dissolve in DMSO.
Hey 88man. Thanks bro. I actually do have a few questions if you don’t mind. After this Sunday coming up, I have one more week on dht gel.
My plan was to use finasteride for 1 month during my break from dht. Finasteride blocks dht in the body and is use for treating male pattern baldness through blocking dht.
After skyping with coughroniellecough strongly advised me to cycle finasteride to block my dht levels for one month. This will increase androgen sensitivity since its being starved of dht for 4 weeks. Then after 4 weeks, Im to hit the dht gel but with extremely high androgen sensitivity from having my dht blocked gains are suppose to skyrocket rapidly during the dht cycle.
Here’s a link to the original Penis Enlargement patent talking about cycling finasteride with dht with your Penis Enlargement routine.
http://www.chemicalenlargement.com/cpereport/cpe1.pdf
Coughroniellecough cycled DHT and finasteride 3 times and he had to stop because his girth reached 7 inches in thickness being to big for most of his clients to handle.
So here’s my question 88man. After reading about finasteride, Im coming across a lot of horror stories on men using it to restore there hair but are reporting permanent libido loss, erectile dysfunction and depression. After reading up on dht blocking hair restoration drugs like finasteride Im leaning towards not doing it in fear of permanently messing up my hormones.
So what Im looking for a safer way to block dht for 4 weeks instead of taking finasteride aka proscar and other dht blocking drugs. So far the only natural supplement that I’ve read on that blocks dht is saw palmetto.
Do you know anything about this 88man. Thanks in advance bro.
Hey MikeShlort and neognostic. I agree. I definitely don’t want any newbies diving into what Im doing without the proper knowledge and mess them selves up.
Ok fellas. Yesterday my injection cocktail was 30 mcgs of igf-des, 100mcgs of tb-500 and my all time high of 57 mcgs of PGE1. The pain of my erection was excruciating the most painful experience I’ve experienced so far. I managed to keep my cable clamp aka cockring on for 40 minutes before taking it off to ensure full pge1 saturation. While wearing the cockring my erection was swollen with red glans and extreme girth ocompanied with extreme throbbing pain that had me in agony.
After taking off the cockring/cable clamp with silicone sleeve, my erection died down to 60 to 70% for 20 minutes then it died down to 40% which totally sucked because even at 40% erect, I was in painful agony as the pge1 circulated through my penis. I was determined to get 2 hours of erection time so I painfully edged to stay at an 70 to 80% erection level.
Im thinking the extreme pain is what’s causing my penis to not stay over 50% erect after taking off the cockring because there were times that when I tried to mentally block out the pain and relax and my chemical erection would sprout back up to 70% without any physical stimulation.
So this evening my dosage will increase yet again hoping for that 2 to 4 hour erection. This evening’s pge1 dosage will be 64 mcgs of liquid PAIN. Wish me luck later on today fellas. Im going to need it. This shit sucks.
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Hey Snakes. Thanks for the response.
If you ever want to try Helichrysum Italicum, give me a pm if you can't find a good cheap source (found one for $28(5ml which = around 120-130 drops) w/ free shipping).
There seems to be 2 types of Italicum, both offering the same thing in terms of scar tissue+ETC skin benefits, but the higher priced
one from croatia($11+ more per 5ml) has pain benefits as well (via Neryl Acetate, benefits for muscle spasms).
I did a search on molecular weights and came up with.
Nattokinase = 27-30k daltons.
Bromelian = 33-35k daltons.
However, I found slight contradictory weights via http://www.peyroniesforum.net/index.php?topic=39.100 , which isn't much off compared to what I found above.
You might look over that thread if you haven't. I haven't just yet, but will check it out once I get time.
Here is another I was looking at as well: Low Molecular HA: http://www.bulkactives.com/ulmwnah.htm (would be VERY potent mixed with DMSO it seems)
Lotion crafters also has it, but somewhat more expensive. They also have a heap of other topical agents they sell as well.
If you ever want to try Helichrysum Italicum, give me a pm if you can't find a good cheap source (found one for $28(5ml which = around 120-130 drops) w/ free shipping).
There seems to be 2 types of Italicum, both offering the same thing in terms of scar tissue+ETC skin benefits, but the higher priced
one from croatia($11+ more per 5ml) has pain benefits as well (via Neryl Acetate, benefits for muscle spasms).
I did a search on molecular weights and came up with.
Nattokinase = 27-30k daltons.
Bromelian = 33-35k daltons.
However, I found slight contradictory weights via http://www.peyroniesforum.net/index.php?topic=39.100 , which isn't much off compared to what I found above.
You might look over that thread if you haven't. I haven't just yet, but will check it out once I get time.
Here is another I was looking at as well: Low Molecular HA: http://www.bulkactives.com/ulmwnah.htm (would be VERY potent mixed with DMSO it seems)
Lotion crafters also has it, but somewhat more expensive. They also have a heap of other topical agents they sell as well.
It's amazing reading this stuff. Still on record as saying, at the end of the day, a protocol will be discovered with virtually ZERO pain.
Also notice how dynamic this process is. In the course of just one thread, Snake has altered his proposed regimen at least THREE times....Add Finasteride...Cycle Finasteride...No Finasteride! lol
That's the trial and error process that Snake's willing to be the guinea pig for. Much thanks.
As for me, I'm grabbing my popcorn, learning and waiting until the end of this chapter is written...
Also notice how dynamic this process is. In the course of just one thread, Snake has altered his proposed regimen at least THREE times....Add Finasteride...Cycle Finasteride...No Finasteride! lol
That's the trial and error process that Snake's willing to be the guinea pig for. Much thanks.
As for me, I'm grabbing my popcorn, learning and waiting until the end of this chapter is written...
Hey smerc. Thanks for the info bro. From the looks of the molecular weight they have stated for Nattokinase and Bromelian on the peyronies source, Nattokinase can be carried through skin with DMSO but bromelian is to large to be carried through. Oh well. Guess I’ll just have to take Bromelian orally like normal people. LOL.
Hey bigdaddy777. I hear you on finding a painless protocol because these pge1 erection workouts are pure torture once dosages get high. Yeah with my constant change in my routine or plans, this is because Im always learning new things with my constant research on finding the best tools to help with my protocol. Cycling finestride with DHT month to month is a great idea to increase androgen sensitivity but Im reading to many horror stories abouth guys taking finestride aka procebia to stop male pattern baldness but are reporting long term erectile dysfunction and libido loss.
Like you said. My whole experiment is all about trial and error. So far I believe that just simply using pge1 in high dosages is all that’s needed for enlargement over the course of 1 to 3 years but the extremely painful erections associated with high pge1 dosages is the reason why I continue to find other path ways of Penis Enlargement to assist with my pge1 erection workouts.
So far my plan once I get off of dht gel is to load up on saw palmetto during my off month since it blocks dht. Im hoping that this will up regulated my androgen sensitivity so when I start my next dht cycle, it will be much more effective.
Also will be adding vasodilator phentolamine and vasodilator forskolin to my injection cocktail. Both of these compounds create a strong pain free erection all by them selves which is ideal for having sex with rather then PAINFUL pge1. Unfortunately PGE1 is still needed because of its collagen delinking properties to the tunica which is the core of my chemical protocol. So by injecting phentolamine and forskolin with pge1, less pge1 will be needed to create a strong 3 to 4 hour erection which also means less pain, thus allowing me to build my pain tolerance up as pge1 dosage goes up slower.
This Thursday that past which was February 28, 2013. For my erection workout session I pinned and all time high of 64 mcgs of PGE1, 100 mcgs of tb-500 and 60 mcgs of MGF. This time I only had the cockring on for 10 minutes. After taking off the cockring, the erection quickly built up with immediate pain. Erection stayed at 60 to 70% without any stimulation for 30 minutes. Pain was extreme.
After 30 minutes my erection died down to 40% in which I had to edge to stay at 60 to 70% erect for another 30 minutes making 1 hour of erection time past 60%.
For the second hour my dick stayed at about 50% erection without any stimulation.
For 3 more hours my flaccid stayed plumped from the workout with some pge1 soreness.
My post pge1 flaccid is nice and thick but the actual erection time is still lacking and well below the 2 to 4 hour mark Im looking for. Will have to go up even though the pain is severe.
Hey bigdaddy777. I hear you on finding a painless protocol because these pge1 erection workouts are pure torture once dosages get high. Yeah with my constant change in my routine or plans, this is because Im always learning new things with my constant research on finding the best tools to help with my protocol. Cycling finestride with DHT month to month is a great idea to increase androgen sensitivity but Im reading to many horror stories abouth guys taking finestride aka procebia to stop male pattern baldness but are reporting long term erectile dysfunction and libido loss.
Like you said. My whole experiment is all about trial and error. So far I believe that just simply using pge1 in high dosages is all that’s needed for enlargement over the course of 1 to 3 years but the extremely painful erections associated with high pge1 dosages is the reason why I continue to find other path ways of Penis Enlargement to assist with my pge1 erection workouts.
So far my plan once I get off of dht gel is to load up on saw palmetto during my off month since it blocks dht. Im hoping that this will up regulated my androgen sensitivity so when I start my next dht cycle, it will be much more effective.
Also will be adding vasodilator phentolamine and vasodilator forskolin to my injection cocktail. Both of these compounds create a strong pain free erection all by them selves which is ideal for having sex with rather then PAINFUL pge1. Unfortunately PGE1 is still needed because of its collagen delinking properties to the tunica which is the core of my chemical protocol. So by injecting phentolamine and forskolin with pge1, less pge1 will be needed to create a strong 3 to 4 hour erection which also means less pain, thus allowing me to build my pain tolerance up as pge1 dosage goes up slower.
This Thursday that past which was February 28, 2013. For my erection workout session I pinned and all time high of 64 mcgs of PGE1, 100 mcgs of tb-500 and 60 mcgs of MGF. This time I only had the cockring on for 10 minutes. After taking off the cockring, the erection quickly built up with immediate pain. Erection stayed at 60 to 70% without any stimulation for 30 minutes. Pain was extreme.
After 30 minutes my erection died down to 40% in which I had to edge to stay at 60 to 70% erect for another 30 minutes making 1 hour of erection time past 60%.
For the second hour my dick stayed at about 50% erection without any stimulation.
For 3 more hours my flaccid stayed plumped from the workout with some pge1 soreness.
My post pge1 flaccid is nice and thick but the actual erection time is still lacking and well below the 2 to 4 hour mark Im looking for. Will have to go up even though the pain is severe.
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March 2, 2013 Friday
Injected 100mcgs of tb-500, 30 mcgs of igf-des and new highest amount of pge1 being 75 mcgs all into the left chamber of the penis with my cock ring on to trapped chems in the penis. I mashed flaccid penis around to distribute the chems all through out the shaft. 10 minutes later I loosen the clamp to unleash the flood gates. Erection built with in 5 minutes accompanied with excruciating pain.
I tighten back the cockring aka silicone sleeve and cable clamp to continue trapping the chems in my penis and keep the erection going. Cable clamp stayed on for 40 minutes. Pain was extreme but I tried to block it out by taking a bath and lying in my tub doing meditation exercises. During the 40 minutes of wearing my cockring, the amount of engorgement I had was amazing. Clamped midgirth measured at 6.8 inches under pge1 influence.
After 40 grueling minutes I took off the cockring but then was bomb rushed with the most extreme pain ever. Erection deflated down to 40% with throbbing pain. Painful erection stayed at 40% for 40 minutes then surprisingly sprouted back to 70 to 75% hardness. After getting back up to 75% erection level, it went back down 10 minutes later to 40% erection then fluctuated for the next 2 hours between 40% and 60% erect.
I think what’s going on is my dick is in so much pain, agony and inflammation that even under the influence of pge1's vasodiolating effects, the immense throbbing pge1 pain makes my penis turtle some, keeping me from reaching and staying above 60% erection level.
I could be wrong but I’m thinking 75mcgs is enough but the intense pain is causing my dick to hide from a full chemical erection. This was the most painful pge1 workout yet with my jump to 75mcgs. After taking my cockring off and getting out of the tub, I was literally walking around my apartment back and forth screaming and cursing in agony with my dick fluctuating between 40% and 60% hardness most of the time.
Like I said before. I have no freaking idea how coughroniellecough was able to with stand the pain of 500 mcgs of pge1 once he built up to it. Then to stack with that the crazy guy was stacking papaverine with it which brings the pain up 10 fold. 75 mcgs is making me squil like a girl. I can only imagine the type of tunica delinking that 500 mcgs of pge1 is doing when the penis is saturated with that amount but the pain is very discouraging. I guess with such high dosages of tunica delinking pge1, that explains coughroniellecough’s gains. A lot of pain and suffering to reach it.
I actually have a headache today from the pain and stress of yesterday’s erection workout but my flaccid is definitely much fatter. Post pge1 demensions, mainly girth is rediculous as pge1 dosage increases.
So my next erection workout is tomorrow in which I’m upping the dosage to 80 or maybe 82 mcgs of pge1. Maybe I should add morphine to my injection cocktail fellas. LOL.
Injected 100mcgs of tb-500, 30 mcgs of igf-des and new highest amount of pge1 being 75 mcgs all into the left chamber of the penis with my cock ring on to trapped chems in the penis. I mashed flaccid penis around to distribute the chems all through out the shaft. 10 minutes later I loosen the clamp to unleash the flood gates. Erection built with in 5 minutes accompanied with excruciating pain.
I tighten back the cockring aka silicone sleeve and cable clamp to continue trapping the chems in my penis and keep the erection going. Cable clamp stayed on for 40 minutes. Pain was extreme but I tried to block it out by taking a bath and lying in my tub doing meditation exercises. During the 40 minutes of wearing my cockring, the amount of engorgement I had was amazing. Clamped midgirth measured at 6.8 inches under pge1 influence.
After 40 grueling minutes I took off the cockring but then was bomb rushed with the most extreme pain ever. Erection deflated down to 40% with throbbing pain. Painful erection stayed at 40% for 40 minutes then surprisingly sprouted back to 70 to 75% hardness. After getting back up to 75% erection level, it went back down 10 minutes later to 40% erection then fluctuated for the next 2 hours between 40% and 60% erect.
I think what’s going on is my dick is in so much pain, agony and inflammation that even under the influence of pge1's vasodiolating effects, the immense throbbing pge1 pain makes my penis turtle some, keeping me from reaching and staying above 60% erection level.
I could be wrong but I’m thinking 75mcgs is enough but the intense pain is causing my dick to hide from a full chemical erection. This was the most painful pge1 workout yet with my jump to 75mcgs. After taking my cockring off and getting out of the tub, I was literally walking around my apartment back and forth screaming and cursing in agony with my dick fluctuating between 40% and 60% hardness most of the time.
Like I said before. I have no freaking idea how coughroniellecough was able to with stand the pain of 500 mcgs of pge1 once he built up to it. Then to stack with that the crazy guy was stacking papaverine with it which brings the pain up 10 fold. 75 mcgs is making me squil like a girl. I can only imagine the type of tunica delinking that 500 mcgs of pge1 is doing when the penis is saturated with that amount but the pain is very discouraging. I guess with such high dosages of tunica delinking pge1, that explains coughroniellecough’s gains. A lot of pain and suffering to reach it.
I actually have a headache today from the pain and stress of yesterday’s erection workout but my flaccid is definitely much fatter. Post pge1 demensions, mainly girth is rediculous as pge1 dosage increases.
So my next erection workout is tomorrow in which I’m upping the dosage to 80 or maybe 82 mcgs of pge1. Maybe I should add morphine to my injection cocktail fellas. LOL.
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Snake when I did chem Penis Enlargement years ago I had the same pain you speak of but quit due to not being able to get enough Pge-1,I used a couple of times a numbing spray or a prolong spray on my dick to help with the pain it helps a little.
Hey Hepcat. When I say turtling I mean that during the pge1 workout the throbbing pain of pge1 keeps my penis from staying above 60% erect at times.
Hey 8incyclops. Yeah I’ve tried the prolonged numbing spray on my dick before injecting. It does seem to numb the pain of the needle going in but the actual erection under pge1 is still extremely painful. I don’t think that spray is penetrating deep into the tunica but instead just numbing out the skin.
This Sunday that just past being March 3, 2013, I injected my usual 100mcgs of tb-500, 75 mcgs of mgf and my new highest amount of pge1 being 80 mcgs into the right chamber of my penis. I only kept my cockring on for 10 minutes after injecting to trap the chems in the shaft giving them time to bind to receptors. After taking off the cockring, the flood gates opened up and erection built with that same ol excruciating pge1 pain. For the first hour I got extremely painful wood fluctuating from 50 to 80% hardness. 2nd hour was 30 to 60% hardness and the 3rd hour was 30 to 40% hardness.
Still not satisfied with the erection quality and duration so I’ll continue to titrate up with PGE1 and face more pain.
One thing that’s starting to bother me now is my EQ is starting to slack with my normal non pge1 erections during sex. Im not reaching full erection and when I do, Im having trouble holding it. A couple of things could be happening here. Maybe the high dosages of pge1 and the extreme pain is burning out my nervous system or my penis is extremely fatigued from the high pge1 dosages and all of the pain and inflammation it’s causing.
Because after a painful pge1 erection workout my dick has some fluid build up like after a heavy pumping set or clamping set. Not sure if a break is in order from overtraining or what. A little bit disappointed with this because the DHT gel is suppose to be keeping my libido high and normal erections on point.
Perhaps supplementing with cialas or goldreallas pills 3 times a week is in order to keep my natural erections up to par from these grueling pge1 workouts.
Hey 8incyclops. Yeah I’ve tried the prolonged numbing spray on my dick before injecting. It does seem to numb the pain of the needle going in but the actual erection under pge1 is still extremely painful. I don’t think that spray is penetrating deep into the tunica but instead just numbing out the skin.
This Sunday that just past being March 3, 2013, I injected my usual 100mcgs of tb-500, 75 mcgs of mgf and my new highest amount of pge1 being 80 mcgs into the right chamber of my penis. I only kept my cockring on for 10 minutes after injecting to trap the chems in the shaft giving them time to bind to receptors. After taking off the cockring, the flood gates opened up and erection built with that same ol excruciating pge1 pain. For the first hour I got extremely painful wood fluctuating from 50 to 80% hardness. 2nd hour was 30 to 60% hardness and the 3rd hour was 30 to 40% hardness.
Still not satisfied with the erection quality and duration so I’ll continue to titrate up with PGE1 and face more pain.
One thing that’s starting to bother me now is my EQ is starting to slack with my normal non pge1 erections during sex. Im not reaching full erection and when I do, Im having trouble holding it. A couple of things could be happening here. Maybe the high dosages of pge1 and the extreme pain is burning out my nervous system or my penis is extremely fatigued from the high pge1 dosages and all of the pain and inflammation it’s causing.
Because after a painful pge1 erection workout my dick has some fluid build up like after a heavy pumping set or clamping set. Not sure if a break is in order from overtraining or what. A little bit disappointed with this because the DHT gel is suppose to be keeping my libido high and normal erections on point.
Perhaps supplementing with cialas or goldreallas pills 3 times a week is in order to keep my natural erections up to par from these grueling pge1 workouts.
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Seun;536751 said:
Not to mention he takes it up the ass
Hey fellas. Yeah Ronielle is gay but who cares. Im interested only in his knowledge and experiance with chemical pe since he's made awsome gains with it. His other extraciricular actvitities I could give two shits about. With him being gay, the way I look at it is more women for me with less competition. LOL.
So yesterday I injected only 15 mcgs of pge1 to fuck one of my regulars being Kara and then my next door neigbor Heather. I injected at 8pm and had Kara over at 8:15pm and fucked the shit out of her. Actually made her bleed from going to deep but she wanted to challenge herself and was determined to take all bpel 9.8 inches of me. Didn't happen. LOL.
Anyway after busting 2 nuts with her, she left and I cleaned up and had my neighbor Heather over to receive her SNAKE PLUGGING. After making her cum 3 times, I busted my 3rd nut of the day and was still rock hard.
This shows just how effective pge1 is to my body when injected at night time versus any other time of the day. It was amazing how 15 mcgs of pge1 granted me 2 to 3 hours of almost painless wood when in the day time, Im up to injecting 80 plus mcgs of pge1 an still struggling to hit the 2 to 4 hour erection mark with undescribable pain in the process.
I wonder why pge1 works so much better at night time close to bed to where considerably less is needed? My only answer is because close to bed time the body is more relaxed. If anybody has any clues to this phenomenon please chime in.
Later on today Im injecting 85 PAINFUL mcgs of PGE1 for this afternoon's chemical erection workout. This is also the last week on my dht cycle which will end this Sunday. Still thinking about taking saw palmetto and other herbs to block dht during the next 4 weeks before I cycle back on dht again. I want my andgregen receptors as sensetive as possible for my next DHT cycle. Even though Finastride has horrible side effects Im still looking at it as an option. Guess Im just crazy like that. LOL.
For all newbies to the Penis Enlargement game. I don't recommend you go the chemical Penis Enlargement route until you max out all of your normal Penis Enlargement gains and if you do decide to go the chemical route please learn and study up as much as you can on the subject before you just dive right in.
So yesterday I injected only 15 mcgs of pge1 to fuck one of my regulars being Kara and then my next door neigbor Heather. I injected at 8pm and had Kara over at 8:15pm and fucked the shit out of her. Actually made her bleed from going to deep but she wanted to challenge herself and was determined to take all bpel 9.8 inches of me. Didn't happen. LOL.
Anyway after busting 2 nuts with her, she left and I cleaned up and had my neighbor Heather over to receive her SNAKE PLUGGING. After making her cum 3 times, I busted my 3rd nut of the day and was still rock hard.
This shows just how effective pge1 is to my body when injected at night time versus any other time of the day. It was amazing how 15 mcgs of pge1 granted me 2 to 3 hours of almost painless wood when in the day time, Im up to injecting 80 plus mcgs of pge1 an still struggling to hit the 2 to 4 hour erection mark with undescribable pain in the process.
I wonder why pge1 works so much better at night time close to bed to where considerably less is needed? My only answer is because close to bed time the body is more relaxed. If anybody has any clues to this phenomenon please chime in.
Later on today Im injecting 85 PAINFUL mcgs of PGE1 for this afternoon's chemical erection workout. This is also the last week on my dht cycle which will end this Sunday. Still thinking about taking saw palmetto and other herbs to block dht during the next 4 weeks before I cycle back on dht again. I want my andgregen receptors as sensetive as possible for my next DHT cycle. Even though Finastride has horrible side effects Im still looking at it as an option. Guess Im just crazy like that. LOL.
For all newbies to the Penis Enlargement game. I don't recommend you go the chemical Penis Enlargement route until you max out all of your normal Penis Enlargement gains and if you do decide to go the chemical route please learn and study up as much as you can on the subject before you just dive right in.
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Lol I feel the same way about it king, more women who haven't had a 7" girth dick for me. I just made that comment to be funny/a prick.
Hey hepcat and MikeShlort. It's cool yall. LOL.
Ok so yesterday March 6 2012. I injected my normal 100 mcgs of tb-500, 60 to 70 mcgs of MGF and my new all time high of 85 mcgs of PGE1. After injecting I kept the cockring on for 10 minutes to trap the chems in the shaft letting them have time to bind to receptors before openning the cockring and unleashing the flood gates. After taking off the cockring my erection immediately built up with the same ol unbearable pain. Got up to an 75 to 80% erection that lasted for 30 minutes. After the first 30 minutes my erection went down to 40% hardness and fluacted between 40% and 60% hardness for 2 more hours. Definietely disappointed with the piss poor erection quality while taking pge1 during the day vs night time close to bed.
My next workout the plan is to inject 90 mcgs of pge1 but now with the use of so much PGE1, Im at a point to where I cannot fit all of my chems into a 1 CC syringe if I go any higher in dosage. I either need to find bigger syringes that's capable of holding more then 1 cc or find a source for a higer concentration of pge1. I'll be ordering my other vasodiolaters tomorrow being phentolamine and for Forskolin to help assist and work in synergy with pge1.
Ok so yesterday March 6 2012. I injected my normal 100 mcgs of tb-500, 60 to 70 mcgs of MGF and my new all time high of 85 mcgs of PGE1. After injecting I kept the cockring on for 10 minutes to trap the chems in the shaft letting them have time to bind to receptors before openning the cockring and unleashing the flood gates. After taking off the cockring my erection immediately built up with the same ol unbearable pain. Got up to an 75 to 80% erection that lasted for 30 minutes. After the first 30 minutes my erection went down to 40% hardness and fluacted between 40% and 60% hardness for 2 more hours. Definietely disappointed with the piss poor erection quality while taking pge1 during the day vs night time close to bed.
My next workout the plan is to inject 90 mcgs of pge1 but now with the use of so much PGE1, Im at a point to where I cannot fit all of my chems into a 1 CC syringe if I go any higher in dosage. I either need to find bigger syringes that's capable of holding more then 1 cc or find a source for a higer concentration of pge1. I'll be ordering my other vasodiolaters tomorrow being phentolamine and for Forskolin to help assist and work in synergy with pge1.
Hey kingsnake,
why do you use PGE1 during the day when the effects are not nearly as good as during evening/night workout, do you think all day tunica delinking is necessary?
why do you use PGE1 during the day when the effects are not nearly as good as during evening/night workout, do you think all day tunica delinking is necessary?
Hey thebrightestday. The reason why I do my erection workouts in the afternoon as oppose to night time is because the theory that the more pge1 used, the more tunica delinking to the tunica. It is already proven that pge1 does indeed remodel and delink collagen in the tunica. At night time my erection quality on pge1 is indeed much better but the down side is much less pge1 is needed which means I could be missing out on optimal tunica delinking. It takes about 10 to 20 mcgs of pge1 to give me a 2 to 4 hour boner if injected close to bedtime. Right now my training dose during the day is 90 mcgs and still climbing. If I inject 90 mcgs at night I’ll have an extremely painful aching boner that will keep me up all night and I’ll get no sleep.
Now with all that was just said. Here’s the million dollar question. Is PGE1 really the key factor in chemical penis enlargement or is simply having a raging tissue expanding boner for 2 to 4 hours at a time the key factor in enlargement.
If you look at Doctor Adam’s chemical Penis Enlargement Patent, his test subjects were being injected with multiple vasodilators and not just pge1. Most of the test subjects were being injected with pge1, papaverine, phentolamine, Atropine, Chlorpromazine.
In the patent here’s TEST SUBJECT or Example 4’s data and results in the notes.
[0086]A male patient, age 34, was treated with intracavernosal injections of a triple mix of the vasodilators Atropine, Chlorpromazine and Papavarine on a regular basis (approximately two to five times per week) over a 4-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of 60-90% over a period of about 3 to 4.5 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator, Potaba®--potassium aminobenzoate (1000 mg/3-4 times per day) was administered orally starting 1 month before starting the IC injections of the vasodilators Atropine, Chlorpromazine and Papavarine.
[0087]After 5 months of treatment the patient's erect penis increased from 6.0 inches to 7.1 inches (about an 18% increase) in length.
Also here is Example 8’s data and results with pge1 not being in his injection cocktail.
[0093]A male patient, age 47, was treated with intracavernosal injections of a triple mix of the vasodilators Atropine, Chlorpromazine and Papavarine on a regular basis (approximately three to four times per week) over a 6-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of an erectile response between 60-95% over a period of several hours, generally 3 to 4.5 hours. The quantity of medication was adjusted from time to time in accordance with the patient's response, which was monitored initially weekly. After 2 months of treatment subcutaneous injections of testosterone 14-20 mg into the penis were added as an accelerator.
[0094]The size of the patient's fully erect penis increased from 5.2 inches to 6.0 inches in length (about a 15% increase) over the 6 month treatment period.
So as you can see, PGE1 wasn’t even used in Example 4 or Example 8’s erection workouts. So maybe painful high dosages of pge1 isn’t needed for results. Maybe just simply injecting a vasodilator that grants a 2 to 6 hour erection is what’s causing the enlargement through just the simple strain and tension of being erect for that amount of time on a daily basis.
Maybe pge1 and its tunica delinking properties aren’t needed at all. Example 4 and 8 just prooved this. If this is the case then that would be terrific because the other vasodilators like Atropine, Chlorpromazine, phentolamine and papaverine don’t cause any pain with the erection they induce. This would mean that I can comfortable walk around my apartment and do other things while having a raging 2 to 4 hour PAINLESS erection. It would definitely be nice to not be holding my erection in agonizing pain for 2 to 4 hours a day which is mentally exhausting.
Once I get my phentolamine and forskolin in the mail and reconstitute it I’ll be trying these vasodilators by them selves first to see what kind of workout they grant without pge1. Most likely I’ll still be adding pge1 vasodilolater in the cocktail to get some tunica delinking effects but maybe I’ll keep the pge1 dosage to a minimum to keep pge1 pain to a minimum.
It’s a lot of information that I have to decide on. Is pge1’s tunica delinking and remodeling effects needed with chemical pe or is simply injecting any vasodilator that grants a strong 2 to 6 hour erection with potentiators like DMSO/PABA to soften the tunica all that’s needed for enlargement?
Once I get my other vaso’s I’ll find out with time.
Anyway though here’s all 10 of the test subjects with there routines and results if yall are interested. Most of them had pge1 in there cocktails and some didn’t. Here are all 10 test subjects below.
EXAMPLE 1
[0080]A male patient, age 41, was treated with intracavernosal injections of a vasodilator, prostaglandin E1, on a regular basis (approximately four to five times per week) over an 18 month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of an erectile response between 40-75% over a period of several hours, generally 3 to 6 hours. The quantity of medication was adjusted from time to time in accordance with the patient's response, which was monitored at least weekly.
[0081]The size of the patient's fully erect penis increased from 5.8 inches to 8.6 inches in length (about an 48% increase) and 3.7 inches to 5.8 inches in girth (about an 56% increase) over the 18-month treatment period. Following the discontinuation of this treatment, the erect penis length remained stable for two years at over 81/2 inches. Treatment was re-institued combining intracavernosal injections 3-4 times per week of a mixture of testosterone (0.5 mg) and vasodilators with low dose oral Potaba (500-1000 mg) 3-4 times per day. After a short treatment period of 21/2 months, the patient's erect penis was over 9 inches in length, which means he has gained an additional 0.4-0.5 inches in length (about an 6% increase). The total increase in length was therefore about 3.2 inches (about an 55% increase) in length.
EXAMPLE 2
[0082]A male patient, age 30, was treated with intracavernosal injections of the vasodilator on a regular basis (approximately four to five times per week) over a 6-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement over a period of about 3 to 6 hours. The quantity of medication was adjusted in accordance with the patient's response, The potentiator potaba (aminobenzoate) (1000 mg/4 times per day) was administered orally to the patient for the last 60 days of treatment.
[0083]The patient's erect penis increased from 5.6 inches to 7.7 inches (about an 38% increase) in length and 3.2 inches to 5.3 inches (about an 65% increase) in girth over the 6-month treatment period.
EXAMPLE 3
[0084]A male patient, age 52, was treated with separate intracavernosal injections of vasodilators, Papavarine, phentolamine and prostaglandin E1, on a regular basis, selected from treatments of 0 to 4 times per week, over a 7 month treatment period along with daily subcutaneous injections of a prostaglandin F analogue. A sufficient quantity of vasodilator was administered to maintain a prolonged engorgement of an erectile response greater than 70% for 3.5-5 hours duration. The quantity of medication was adjusted from time to time in accordance with the patient's response, which was monitored initially weekly then monthly once the patient had mastered the IC technique and the responses were consistently of the same duration.
[0085]The size of the patient's fully erect penis increased from 5.0 inches to 6.3 inches in length, i.e. about a 26% increase, over the 7-month treatment period. Following the discontinuation of this treatment, the erect penis length remained stable.
EXAMPLE 4
[0086]A male patient, age 34, was treated with intracavernosal injections of a triple mix of the vasodilators Atropine, Chlorpromazine and Papavarine on a regular basis (approximately two to five times per week) over a 4-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of 60-90% over a period of about 3 to 4.5 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator, Potaba®--potassium aminobenzoate (1000 mg/3-4 times per day) was administered orally starting 1 month before starting the IC injections of the vasodilators Atropine, Chlorpromazine and Papavarine.
[0087]After 5 months of treatment the patient's erect penis increased from 6.0 inches to 7.1 inches (about an 18% increase) in length.
EXAMPLE 5
[0088]A male patient, age 44, was treated with intracavernosal injections of a quadruple mix of the vasodilators prostaglandin E1, Atropine, Chlorpromazine and Papavarine on a regular basis (approximately two to four times per week) over a 4-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement over a period of about 3 to 5 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator dihydrotestosterone 5% ointment was administered orally starting two weeks before starting the IC injections of the vasodilators Atropine, Chlorpromazine and Papavarine and prostaglandin.
[0089]After 4 months of treatment the patient's erect penis increased from 5.2 inches to 6.5 inches (about a 25% increase) in length.
EXAMPLE 6
[0090]A male patient, age 44, was treated with intracavernosal injections of the vasodilator phentolamine on a regular basis (approximately two to four times per week) over a 4-month treatment period. Phentolamine was frequently combined with indirect vasodilating effects of oral Viagra to produce and maintain a prolonged engorgement of 60-90% over a period of about 3 to 5 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator dihydrotestosterone gel was administered orally starting two weeks before starting the IC injections of the vasodilators Atropine, Chlorpromazine and Papavarine and prostaglandin.
[0091]After 4 months of treatment the patient's erect penis increased from 5.2 inches to 6.5 inches (about a 25% increase) in length.
EXAMPLE 7
[0092]A male patient, age 72, was treated with intracavernosal injections of the quadruple mix of the vasodilators prostaglandin E1, Atropine, Chlorpromazine and Papavarine on a regular basis (approximately two to four times per week) over a 3-month treatment. The indirect vasodilating effects of oral Cialis and Levitra were sometimes added to the quadruple mix of the vasodilators prostaglandin E1, Atropine, Chlorpromazine and Papavarine to produce and maintain a prolonged engorgement of 60-85% over a period of about 2.5 to 3 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiators Potaba 1000 mg 4×/day orally and prostaglandin F topically were also used with the vasodilators. After 3 months of treatment the patient's erect penis increased from 6.5 inches to 7.1 inches (about a 9% increase) in length.
EXAMPLE 8
[0093]A male patient, age 47, was treated with intracavernosal injections of a triple mix of the vasodilators Atropine, Chlorpromazine and Papavarine on a regular basis (approximately three to four times per week) over a 6-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of an erectile response between 60-95% over a period of several hours, generally 3 to 4.5 hours. The quantity of medication was adjusted from time to time in accordance with the patient's response, which was monitored initially weekly. After 2 months of treatment subcutaneous injections of testosterone 14-20 mg into the penis were added as an accelerator.
[0094]The size of the patient's fully erect penis increased from 5.2 inches to 6.0 inches in length (about a 15% increase) over the 6 month treatment period.
EXAMPLE 9
[0095]A male patient, age 52, was treated with intracavernosal injections of the quadruple mix of the vasodilators prostaglandin E1, Atropine, Phentolamine and Papavarine on a regular basis (using IC medications approximately two to four days per week) over a 3-month treatment. Since the maximum duration of the engorgement of the erection from a single dose was only 45 to 80 minutes, the patient used two to three separate IC injects spaced through out the treatment days to achieve a total i.e. cumulative daily duration of 3 to 4 hours. The indirect vasodilating effects of oral Cialis and Levitra were sometimes added to the quadruple mix of the vasodilators prostaglandin E1, Atropine, Chlorpromazine and phentolamine to produce and maintain a prolonged engorgement of 60-85% over a period of about 3 to 4 hours.
[0096]The quantity of medication was adjusted in accordance with the patient's response. The potentiator Potaba 1000mg 4×/day orally was used with the vasodilator. After 4 months of treatment the patient's erect penis increased from 5.4 inches to 6.1 inches (about a 13% increase) in length and 4.4 to 5.1 inches in circumference (about a 16% increase in circumference).
EXAMPLE 10
[0097]A male patient, age 27, was treated with intracavernosal injections of a prostaglandin E1 on a regular basis (approximately two to five times per week) over a 3-month treatment period. Due to a sensitivity to Prostraglandin E1 causing aching and pain at higher doses, the maximum tolerated dose which produced a comfortable erection was only lasting 90 to 120 minutes. The patient used two separate IC injects spaced throughout the treatment days to achieve a total daily cumulative engorgement duration of 3 to 4 hours. The quantity of medication was adjusted in accordance with the patient's response. The 15 mg of the potentiator Dihydrotestosterone was injected subcutaenously into the penis daily throughout the treatment period. After 3 months of treatment the patient's erect penis increased from 6.3 inches to 7.1 inches (about an 13% increase) in length.
[0098]Although various examples of combined elements of the invention have been described, it will also be understood that these are not intended to be exhaustive and features of one embodiment may be combined with those of another, and such other combinations are contemplated to be within the scope of the invention disclosed herein.
[0099]All publications and other documents mentioned herein are hereby incorporated by reference into this specification.
[0100]While preferred embodiments of the invention have been illustrated and described, it will be appreciated that various changes and modifications can be made therein without departing from the spirit and scope of the invention as defined by the following claims.
Example 10 is funny to me because he actually complained about the pain of pge1 erections in high doses which I totally agree with so the doctors had him do 2 separate injection pge1 erection workouts in smaller dosages to compensate but still get the necessary erection time in.
Here’s the link to the patent as well.
http://www.faqs.org/patents/app/20100112027
Now with all that was just said. Here’s the million dollar question. Is PGE1 really the key factor in chemical penis enlargement or is simply having a raging tissue expanding boner for 2 to 4 hours at a time the key factor in enlargement.
If you look at Doctor Adam’s chemical Penis Enlargement Patent, his test subjects were being injected with multiple vasodilators and not just pge1. Most of the test subjects were being injected with pge1, papaverine, phentolamine, Atropine, Chlorpromazine.
In the patent here’s TEST SUBJECT or Example 4’s data and results in the notes.
[0086]A male patient, age 34, was treated with intracavernosal injections of a triple mix of the vasodilators Atropine, Chlorpromazine and Papavarine on a regular basis (approximately two to five times per week) over a 4-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of 60-90% over a period of about 3 to 4.5 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator, Potaba®--potassium aminobenzoate (1000 mg/3-4 times per day) was administered orally starting 1 month before starting the IC injections of the vasodilators Atropine, Chlorpromazine and Papavarine.
[0087]After 5 months of treatment the patient's erect penis increased from 6.0 inches to 7.1 inches (about an 18% increase) in length.
Also here is Example 8’s data and results with pge1 not being in his injection cocktail.
[0093]A male patient, age 47, was treated with intracavernosal injections of a triple mix of the vasodilators Atropine, Chlorpromazine and Papavarine on a regular basis (approximately three to four times per week) over a 6-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of an erectile response between 60-95% over a period of several hours, generally 3 to 4.5 hours. The quantity of medication was adjusted from time to time in accordance with the patient's response, which was monitored initially weekly. After 2 months of treatment subcutaneous injections of testosterone 14-20 mg into the penis were added as an accelerator.
[0094]The size of the patient's fully erect penis increased from 5.2 inches to 6.0 inches in length (about a 15% increase) over the 6 month treatment period.
So as you can see, PGE1 wasn’t even used in Example 4 or Example 8’s erection workouts. So maybe painful high dosages of pge1 isn’t needed for results. Maybe just simply injecting a vasodilator that grants a 2 to 6 hour erection is what’s causing the enlargement through just the simple strain and tension of being erect for that amount of time on a daily basis.
Maybe pge1 and its tunica delinking properties aren’t needed at all. Example 4 and 8 just prooved this. If this is the case then that would be terrific because the other vasodilators like Atropine, Chlorpromazine, phentolamine and papaverine don’t cause any pain with the erection they induce. This would mean that I can comfortable walk around my apartment and do other things while having a raging 2 to 4 hour PAINLESS erection. It would definitely be nice to not be holding my erection in agonizing pain for 2 to 4 hours a day which is mentally exhausting.
Once I get my phentolamine and forskolin in the mail and reconstitute it I’ll be trying these vasodilators by them selves first to see what kind of workout they grant without pge1. Most likely I’ll still be adding pge1 vasodilolater in the cocktail to get some tunica delinking effects but maybe I’ll keep the pge1 dosage to a minimum to keep pge1 pain to a minimum.
It’s a lot of information that I have to decide on. Is pge1’s tunica delinking and remodeling effects needed with chemical pe or is simply injecting any vasodilator that grants a strong 2 to 6 hour erection with potentiators like DMSO/PABA to soften the tunica all that’s needed for enlargement?
Once I get my other vaso’s I’ll find out with time.
Anyway though here’s all 10 of the test subjects with there routines and results if yall are interested. Most of them had pge1 in there cocktails and some didn’t. Here are all 10 test subjects below.
EXAMPLE 1
[0080]A male patient, age 41, was treated with intracavernosal injections of a vasodilator, prostaglandin E1, on a regular basis (approximately four to five times per week) over an 18 month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of an erectile response between 40-75% over a period of several hours, generally 3 to 6 hours. The quantity of medication was adjusted from time to time in accordance with the patient's response, which was monitored at least weekly.
[0081]The size of the patient's fully erect penis increased from 5.8 inches to 8.6 inches in length (about an 48% increase) and 3.7 inches to 5.8 inches in girth (about an 56% increase) over the 18-month treatment period. Following the discontinuation of this treatment, the erect penis length remained stable for two years at over 81/2 inches. Treatment was re-institued combining intracavernosal injections 3-4 times per week of a mixture of testosterone (0.5 mg) and vasodilators with low dose oral Potaba (500-1000 mg) 3-4 times per day. After a short treatment period of 21/2 months, the patient's erect penis was over 9 inches in length, which means he has gained an additional 0.4-0.5 inches in length (about an 6% increase). The total increase in length was therefore about 3.2 inches (about an 55% increase) in length.
EXAMPLE 2
[0082]A male patient, age 30, was treated with intracavernosal injections of the vasodilator on a regular basis (approximately four to five times per week) over a 6-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement over a period of about 3 to 6 hours. The quantity of medication was adjusted in accordance with the patient's response, The potentiator potaba (aminobenzoate) (1000 mg/4 times per day) was administered orally to the patient for the last 60 days of treatment.
[0083]The patient's erect penis increased from 5.6 inches to 7.7 inches (about an 38% increase) in length and 3.2 inches to 5.3 inches (about an 65% increase) in girth over the 6-month treatment period.
EXAMPLE 3
[0084]A male patient, age 52, was treated with separate intracavernosal injections of vasodilators, Papavarine, phentolamine and prostaglandin E1, on a regular basis, selected from treatments of 0 to 4 times per week, over a 7 month treatment period along with daily subcutaneous injections of a prostaglandin F analogue. A sufficient quantity of vasodilator was administered to maintain a prolonged engorgement of an erectile response greater than 70% for 3.5-5 hours duration. The quantity of medication was adjusted from time to time in accordance with the patient's response, which was monitored initially weekly then monthly once the patient had mastered the IC technique and the responses were consistently of the same duration.
[0085]The size of the patient's fully erect penis increased from 5.0 inches to 6.3 inches in length, i.e. about a 26% increase, over the 7-month treatment period. Following the discontinuation of this treatment, the erect penis length remained stable.
EXAMPLE 4
[0086]A male patient, age 34, was treated with intracavernosal injections of a triple mix of the vasodilators Atropine, Chlorpromazine and Papavarine on a regular basis (approximately two to five times per week) over a 4-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of 60-90% over a period of about 3 to 4.5 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator, Potaba®--potassium aminobenzoate (1000 mg/3-4 times per day) was administered orally starting 1 month before starting the IC injections of the vasodilators Atropine, Chlorpromazine and Papavarine.
[0087]After 5 months of treatment the patient's erect penis increased from 6.0 inches to 7.1 inches (about an 18% increase) in length.
EXAMPLE 5
[0088]A male patient, age 44, was treated with intracavernosal injections of a quadruple mix of the vasodilators prostaglandin E1, Atropine, Chlorpromazine and Papavarine on a regular basis (approximately two to four times per week) over a 4-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement over a period of about 3 to 5 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator dihydrotestosterone 5% ointment was administered orally starting two weeks before starting the IC injections of the vasodilators Atropine, Chlorpromazine and Papavarine and prostaglandin.
[0089]After 4 months of treatment the patient's erect penis increased from 5.2 inches to 6.5 inches (about a 25% increase) in length.
EXAMPLE 6
[0090]A male patient, age 44, was treated with intracavernosal injections of the vasodilator phentolamine on a regular basis (approximately two to four times per week) over a 4-month treatment period. Phentolamine was frequently combined with indirect vasodilating effects of oral Viagra to produce and maintain a prolonged engorgement of 60-90% over a period of about 3 to 5 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator dihydrotestosterone gel was administered orally starting two weeks before starting the IC injections of the vasodilators Atropine, Chlorpromazine and Papavarine and prostaglandin.
[0091]After 4 months of treatment the patient's erect penis increased from 5.2 inches to 6.5 inches (about a 25% increase) in length.
EXAMPLE 7
[0092]A male patient, age 72, was treated with intracavernosal injections of the quadruple mix of the vasodilators prostaglandin E1, Atropine, Chlorpromazine and Papavarine on a regular basis (approximately two to four times per week) over a 3-month treatment. The indirect vasodilating effects of oral Cialis and Levitra were sometimes added to the quadruple mix of the vasodilators prostaglandin E1, Atropine, Chlorpromazine and Papavarine to produce and maintain a prolonged engorgement of 60-85% over a period of about 2.5 to 3 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiators Potaba 1000 mg 4×/day orally and prostaglandin F topically were also used with the vasodilators. After 3 months of treatment the patient's erect penis increased from 6.5 inches to 7.1 inches (about a 9% increase) in length.
EXAMPLE 8
[0093]A male patient, age 47, was treated with intracavernosal injections of a triple mix of the vasodilators Atropine, Chlorpromazine and Papavarine on a regular basis (approximately three to four times per week) over a 6-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of an erectile response between 60-95% over a period of several hours, generally 3 to 4.5 hours. The quantity of medication was adjusted from time to time in accordance with the patient's response, which was monitored initially weekly. After 2 months of treatment subcutaneous injections of testosterone 14-20 mg into the penis were added as an accelerator.
[0094]The size of the patient's fully erect penis increased from 5.2 inches to 6.0 inches in length (about a 15% increase) over the 6 month treatment period.
EXAMPLE 9
[0095]A male patient, age 52, was treated with intracavernosal injections of the quadruple mix of the vasodilators prostaglandin E1, Atropine, Phentolamine and Papavarine on a regular basis (using IC medications approximately two to four days per week) over a 3-month treatment. Since the maximum duration of the engorgement of the erection from a single dose was only 45 to 80 minutes, the patient used two to three separate IC injects spaced through out the treatment days to achieve a total i.e. cumulative daily duration of 3 to 4 hours. The indirect vasodilating effects of oral Cialis and Levitra were sometimes added to the quadruple mix of the vasodilators prostaglandin E1, Atropine, Chlorpromazine and phentolamine to produce and maintain a prolonged engorgement of 60-85% over a period of about 3 to 4 hours.
[0096]The quantity of medication was adjusted in accordance with the patient's response. The potentiator Potaba 1000mg 4×/day orally was used with the vasodilator. After 4 months of treatment the patient's erect penis increased from 5.4 inches to 6.1 inches (about a 13% increase) in length and 4.4 to 5.1 inches in circumference (about a 16% increase in circumference).
EXAMPLE 10
[0097]A male patient, age 27, was treated with intracavernosal injections of a prostaglandin E1 on a regular basis (approximately two to five times per week) over a 3-month treatment period. Due to a sensitivity to Prostraglandin E1 causing aching and pain at higher doses, the maximum tolerated dose which produced a comfortable erection was only lasting 90 to 120 minutes. The patient used two separate IC injects spaced throughout the treatment days to achieve a total daily cumulative engorgement duration of 3 to 4 hours. The quantity of medication was adjusted in accordance with the patient's response. The 15 mg of the potentiator Dihydrotestosterone was injected subcutaenously into the penis daily throughout the treatment period. After 3 months of treatment the patient's erect penis increased from 6.3 inches to 7.1 inches (about an 13% increase) in length.
[0098]Although various examples of combined elements of the invention have been described, it will also be understood that these are not intended to be exhaustive and features of one embodiment may be combined with those of another, and such other combinations are contemplated to be within the scope of the invention disclosed herein.
[0099]All publications and other documents mentioned herein are hereby incorporated by reference into this specification.
[0100]While preferred embodiments of the invention have been illustrated and described, it will be appreciated that various changes and modifications can be made therein without departing from the spirit and scope of the invention as defined by the following claims.
Example 10 is funny to me because he actually complained about the pain of pge1 erections in high doses which I totally agree with so the doctors had him do 2 separate injection pge1 erection workouts in smaller dosages to compensate but still get the necessary erection time in.
Here’s the link to the patent as well.
http://www.faqs.org/patents/app/20100112027
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We need to find out if PGE1 has any chemical properties that makes it a collagen delinker or if it is purely through the tissue expanding properties. It may also be via an indirect mechanism such as increased blood flow (not in relation to the blood causing tissue expansion but an increase in supply of nutrients) or one of PGE1s metabolites.
Ok found this:
http://www.ncbi.nlm.nih.gov/puBathmateed/8533442
"The influence of 13,14-dihydro-PGE1 (PGE0), a biologically active metabolite of PGE1, on collagen and glycosaminoglycan synthesis by the rabbit arterial wall was assessed and compared with the effect of PGE1. Collagen (COL) and glycosaminoglycan (GAG) synthesis was measured using 14C proline- and 35S-incorporation respectively and both were subsequently quantified by autoradiography. PGE1 decreased GAG-synthesis by 40%, while PGE0 caused a 25% decrease. COL-synthesis after PGE1 treatment was diminished by 35%, while the biologically active metabolite caused a drop of 30%. Five and 15 micrograms/kg doses of both compounds were almost equally effective, 1 microgram was without effect. These findings indicate that PGE0 shares the inhibitory effect of PGE1 on COL and GAG biosynthesis. This metabolite has about 60 to 70% of the biological activity of its parent compound PGE1. These results suggest that part of the effects of PGE1 in inhibiting extracellular matrix production could be due to its metabolite PGE0."
http://www.ncbi.nlm.nih.gov/puBathmateed/8533442
"The influence of 13,14-dihydro-PGE1 (PGE0), a biologically active metabolite of PGE1, on collagen and glycosaminoglycan synthesis by the rabbit arterial wall was assessed and compared with the effect of PGE1. Collagen (COL) and glycosaminoglycan (GAG) synthesis was measured using 14C proline- and 35S-incorporation respectively and both were subsequently quantified by autoradiography. PGE1 decreased GAG-synthesis by 40%, while PGE0 caused a 25% decrease. COL-synthesis after PGE1 treatment was diminished by 35%, while the biologically active metabolite caused a drop of 30%. Five and 15 micrograms/kg doses of both compounds were almost equally effective, 1 microgram was without effect. These findings indicate that PGE0 shares the inhibitory effect of PGE1 on COL and GAG biosynthesis. This metabolite has about 60 to 70% of the biological activity of its parent compound PGE1. These results suggest that part of the effects of PGE1 in inhibiting extracellular matrix production could be due to its metabolite PGE0."
That study states that PGE1 and PGE0 cause a decrease in the production of collagen. I'm thinking that maybe PGE1 acts not as a collagen delinker but as a protective agent against the overproduction of collagen. When we Penis Enlargement we run the risk of causing too much collagen to be produced which will hinder future gains but by taking PGE1 we can prevent this.
I wondering what is the best way to take PGE1 - before a Penis Enlargement workout or straight after?
If taken before a workout the PGE1 may no longer have it's collagen production reducing effects acting in the penis when you are doing the workout, however,
taking it after a workout may be too late as you penis may already have started it's collagen production in response to the stress of the workout.
I may be experimenting soon with sub cutaneous injections of PGE1 and tb4 into the penis in order to prevent toughening of the outer layers of the tunica. I will probably try this post hanging or I may be able to get away with doing it immediately before hanging.
I wondering what is the best way to take PGE1 - before a Penis Enlargement workout or straight after?
If taken before a workout the PGE1 may no longer have it's collagen production reducing effects acting in the penis when you are doing the workout, however,
taking it after a workout may be too late as you penis may already have started it's collagen production in response to the stress of the workout.
I may be experimenting soon with sub cutaneous injections of PGE1 and tb4 into the penis in order to prevent toughening of the outer layers of the tunica. I will probably try this post hanging or I may be able to get away with doing it immediately before hanging.
I wish I could have been a part of that study~ heh. Example 1 had dramatic results - bordering on ridiculous and almost false to a casual reader.
Not like I'll have time to experiment until after my time in the military is up. Anyway, king, can you reply to the pm I sent a while back?
Not like I'll have time to experiment until after my time in the military is up. Anyway, king, can you reply to the pm I sent a while back?
kingsnake;536726 said:
why not pump after injection with low pressure/many sets?
kingsnake;537495 said:
probably both. you could even alternate them..
Its the errection time for sure that makes the biggest difference.
Think of the extreme magalophallus cases. They grew after prolonged stress from erections and they didnt supplement with anything.
The key is to not get the tunica toughen up. I think thats also the essence of your tunica article...
IMO..You need to up the intensity, variation or decondition.
Kingsnake, thanks for your continuing extensive research and analysis of chemical Penis Enlargement methods and for keeping the brotherhood informed. As more and more experimenters (chem Penis Enlargement'ers) try various methods, it will be interesting to see which of the methods in Dr. Adam's patent or others prove effective.
Noticed that examples 5 and 6 both indicated that DHT was taken orally, but example 10 indicated subcutaneous injections of DHT. I didn't realize that oral DHT was available, so I presume the reference to oral ingestion was in error especially since in example 6 the DHT was called a "gel".
Noticed that examples 5 and 6 both indicated that DHT was taken orally, but example 10 indicated subcutaneous injections of DHT. I didn't realize that oral DHT was available, so I presume the reference to oral ingestion was in error especially since in example 6 the DHT was called a "gel".
I'm not aware of anyone else who is trying it. It might not do anything but being a scientist I like to experiment.
Kingsnake have you thought about changing up your workouts? I'm thinking that you really need to attack the tunica - lots of hanging, wet jelqing at low erections and lots of [words=https://shop.mattersofsize.com/products/sizegenetics-penis-extender]extender[/words] use. Maybe cut out all girth based exercises.
Kingsnake have you thought about changing up your workouts? I'm thinking that you really need to attack the tunica - lots of hanging, wet jelqing at low erections and lots of [words=https://shop.mattersofsize.com/products/sizegenetics-penis-extender]extender[/words] use. Maybe cut out all girth based exercises.
Seun;537496 said:
I think cycling girth work is necessary. Gain, cement, break, ensure gains are permanent for 3 months at least. Repeat.
I don't know where you got this information and proof that PGE1 "delinks" the tunica. Do you have references you can post, snake? I've not heard anything about this, didn't see it mentioned anywhere in Dr. Adam's work.
And what do you even mean by "delinking" the tunica? It makes the rubber-like sheet of tendon surrounding the CCs weaker and more pliable? Couldn't tunica stretch be achieved from hanging? You can't hang heavy while on PGE1 so what is the purpose?
If this is the theory, then wouldn't you want to pump immediately after the PGE1 erection subsides to take advantage of this "delinking"?
And what do you even mean by "delinking" the tunica? It makes the rubber-like sheet of tendon surrounding the CCs weaker and more pliable? Couldn't tunica stretch be achieved from hanging? You can't hang heavy while on PGE1 so what is the purpose?
If this is the theory, then wouldn't you want to pump immediately after the PGE1 erection subsides to take advantage of this "delinking"?
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bigdex28;537504 said:
I was told by a doctor to keep doses low to avoid inflammation because inflammation leads to collagen production, which leads to tougher to deform tendons etc. Calcium buildup on the tendons. Verapamil is a topical cream which helps block calcium deposits. I was prescribed this after pumping for a few months because the pumpng caused inflammation and the doctor examined my penis and could instantly tell I had a buildup. The verapamil seemed successful, as my dorsal tendon became much less thick and taut in a few months.
I imagine the DMSO essentially does the same thing as Verapamil but not sure if it has any calcium blocking properties.
agent7x6;537519 said:
Do not inject like that. It will cause painful pockets of PGE1 in your penis. I was told this by a doctor. You want it in the CCs and nowhere else. If you aren't in deep enough for the syringe to push down with no resistance, you're doing it wrong.
You people need to seek professional advice on this and stop theorizing. You're going to fuck yourselves up. Why not just try doing actual exercises and using devices properly, daily and committed instead of all this theorizing. Now just using plain old PGE1 isn't enough. Have any of you actually tried just using PGE1 for the 18 months recommended to see the results? No, because that's a lot harder than being a google-search chemist and theorizing. Christ.
Hey Seun. Not sure if a deacon break is needed. I’m thinking what is needed for further progress is for me to find the right dosage to stay above a 75% erection level for 2 to 4 hours. What’s happening right now is pge1 by itself is not keeping me above 75% hardness for over 2 to 4 hours so biomechanical creep can take place as tunica delinking is going on. As soon as I get my other vasodilators to help me get my erection time in, I’m confident that things will improve.
Hey bigdex28. Good find on the info bro.
Hey Syd Kitten. Yeah I know. Example 1 made the biggest gains out of all 10 test subjects. Going from 5 inches to over 9 inches is a huge gain in 18 months time.
Hey dickerschwanz. The thing is I use to pump right after my pge1 erections would subside which is extremely painful do to post pge1 soreness. Now that I’m injecting almost 100 mcgs of pge1 the pain is way too much for me to pump during or right after. Not a chance in hell unless I’m drugged up on strong pain blockers or something. LOL.
I’m am starting to agree with you about chemical Penis Enlargement being the erection time being more important then delinking properties of pge1. If it turns out that largest doses of pge1 isn’t needed and it is indeed the erection time that really matters then other vasodilators can work to achieve a 2 to 5 hour erection without pain.
I could just use 50 mcgs of pge1 which will bring manageable pain then find the right dosage of phentolamine and forsklin to stack with it to bring me up to the 3plus hours of erection at above 75% hardness time mark for better progress.
Hey agent7x6. Yeah examples 5 and 6 did use dht and example 10 was actually getting dht injected into his penis with the vasodilators.
Hey MikeShlort. Here’s a link showing the benefits of pge1 aka prostaglandin.
http://www.ncbi.nlm.nih.gov/puBathmateed/7861547
When I say delinking I mean literally that pge1 unwoven and unravel the cross sectional collagen bonds of the tunica at microscopic levels. This is a process that takes months. As more collagen is unwoven under internal stress which would be the stress of the erection itself and other forms of Penis Enlargement, the tunica will start to give into the stress and enlarge through deformation and mechanical creep. As deformation takes place new cells are generated to fill in the gaps making the tunica sheath that covers the CC and the CS longer which means more blood can engorge the CC and CS before the tunica pressurizes and becomes 100% rigid meaning full erection.
When this happens you have enlargement. Keep repeating this over months to years and the tunica will continue to unweave and remodel giving more and more slack so more blood can engorge the penis before the tunica pressurizes into a full rigid erection. With pge1’s unraveling effects this causes the growth of the tunica through stretch force by expanding the sheath and not the thickness which is what we want because if the thickness of the tunica through cross sectional collagen synthesis builds up then it will make it harder to make gains. AKA TOUGH TUNICA SYNDROME. That’s why the pge1 vasodilator, tb-500 and the DMSO/PABA are used to prevent this.
Here are some quotes in the detailed description of invention in Dr Adam’s patent showing pge1’s aka prostaglandin’s delinking effects. He also talks about the use of relaxin as a potentiator as well.
“0055] Relaxin directly and indirectly triggers a cascade of complex biochemical and cellular effects that can cause general morphological changes to genitalia. Prostaglandins such as prostaglandin F2 alpha and prostaglandin E2 have similar effects. This invention includes the mediators of these cascades as potentiators”.
[0043]A prostaglandin potentiator can be prostaglandin F2 alpha or prostaglandin E2. The potentiator might be relaxin, prostaglandin F2 alpha, or prostaglandin E2, or the biochemical mediators that result in the desired changes in collagen or the connective tissue that produces and remodels collagen and express the effects of relaxin, prostaglandin F2 alpha, or prostaglandin E2.
[0054]The potentiator may be a pharmacological agent or combination of agents that promote cellular processes that result in biological and/or mechanical creep and ultimately induce remodeling of the connective tissues that help define the size and shape of the penis. In addition, an agent which increases solubility of collagen may be used as a potentiator. Agents with very specific mechanisms of action may be used, or other agents with pleomorphic mechanisms of action, such as relaxin or growth hormone which trigger diverse mechanisms to induce growth in the penis may be used. For example, agents may be administered that facilitate the elongation of collagen fibers and accelerate the turnover remodeling rates of collagen through numerous mechanisms. For example, D-penicillamine and dimethyl sulfoxide (DMSO), which promote the elongation of collagen by inhibiting or interfering with inter- and intramolecular collagen cross-linkage may be used. Other agents include, but are not limited to, relaxin, insulin like growth factors, growth hormone, metalloproteinases or metalloproteinases agonists or promoters of collagenase activity, tissue inhibitors of matrix metalloprotenases (TIMPs) other agents that increase collagen solubility, prostaglandins, corticosteroids, or aminobenzoate potassium, a commercial brand being known as Potaba®. Preferred prostaglandins are prostaglandin F2 alpha and prostaglandin E2. Also included are pharmaceutically active sequences, peptidomimetics, or mimetics above the above-listed molecules.
Hey Mikeshlorts. With you saying to just use pge1 for 18 months keep in mind that I’ve been using pge1 now for over 5 months and have reached a point to here I need high dosages to have a good 2 to 4 hour erection if im not injecting close to bedtime. Right now at 90 mcgs it’s not getting me there and the pain is crazy with that much pge1. Not to mention at this point I can’t fit any more pge1 in a 1 CC syringe. I either need to find a source for a higher pge1 concentration or just use other stronger vasodilators to stack with a smaller amount of pge1 to reach 2 to 4 hour erections again like what was done in the patent and what Ronielle did after he reached 500 mcgs of pge1.
Keep in mind that yes there are a lot of theories going around but that’s the point of this thread. What Im doing is experimenting on myself to see what’s the optimal way of aheaving enlargement. So far I’ve gained a quarter inch in midgirth and base girth so this does work even for a vet like me. Right now I’m leaning towards the using what ever safe vasodilator necessary to achieve a nice tunica stretching 2 to 5 hour erection and starting to lean away from the goal of reaching 500 mcgs of PAINFUL pge1 like some of the test subjects did and Ronielle did.
Hey bigdex28. Good find on the info bro.
Hey Syd Kitten. Yeah I know. Example 1 made the biggest gains out of all 10 test subjects. Going from 5 inches to over 9 inches is a huge gain in 18 months time.
Hey dickerschwanz. The thing is I use to pump right after my pge1 erections would subside which is extremely painful do to post pge1 soreness. Now that I’m injecting almost 100 mcgs of pge1 the pain is way too much for me to pump during or right after. Not a chance in hell unless I’m drugged up on strong pain blockers or something. LOL.
I’m am starting to agree with you about chemical Penis Enlargement being the erection time being more important then delinking properties of pge1. If it turns out that largest doses of pge1 isn’t needed and it is indeed the erection time that really matters then other vasodilators can work to achieve a 2 to 5 hour erection without pain.
I could just use 50 mcgs of pge1 which will bring manageable pain then find the right dosage of phentolamine and forsklin to stack with it to bring me up to the 3plus hours of erection at above 75% hardness time mark for better progress.
Hey agent7x6. Yeah examples 5 and 6 did use dht and example 10 was actually getting dht injected into his penis with the vasodilators.
Hey MikeShlort. Here’s a link showing the benefits of pge1 aka prostaglandin.
http://www.ncbi.nlm.nih.gov/puBathmateed/7861547
When I say delinking I mean literally that pge1 unwoven and unravel the cross sectional collagen bonds of the tunica at microscopic levels. This is a process that takes months. As more collagen is unwoven under internal stress which would be the stress of the erection itself and other forms of Penis Enlargement, the tunica will start to give into the stress and enlarge through deformation and mechanical creep. As deformation takes place new cells are generated to fill in the gaps making the tunica sheath that covers the CC and the CS longer which means more blood can engorge the CC and CS before the tunica pressurizes and becomes 100% rigid meaning full erection.
When this happens you have enlargement. Keep repeating this over months to years and the tunica will continue to unweave and remodel giving more and more slack so more blood can engorge the penis before the tunica pressurizes into a full rigid erection. With pge1’s unraveling effects this causes the growth of the tunica through stretch force by expanding the sheath and not the thickness which is what we want because if the thickness of the tunica through cross sectional collagen synthesis builds up then it will make it harder to make gains. AKA TOUGH TUNICA SYNDROME. That’s why the pge1 vasodilator, tb-500 and the DMSO/PABA are used to prevent this.
Here are some quotes in the detailed description of invention in Dr Adam’s patent showing pge1’s aka prostaglandin’s delinking effects. He also talks about the use of relaxin as a potentiator as well.
“0055] Relaxin directly and indirectly triggers a cascade of complex biochemical and cellular effects that can cause general morphological changes to genitalia. Prostaglandins such as prostaglandin F2 alpha and prostaglandin E2 have similar effects. This invention includes the mediators of these cascades as potentiators”.
[0043]A prostaglandin potentiator can be prostaglandin F2 alpha or prostaglandin E2. The potentiator might be relaxin, prostaglandin F2 alpha, or prostaglandin E2, or the biochemical mediators that result in the desired changes in collagen or the connective tissue that produces and remodels collagen and express the effects of relaxin, prostaglandin F2 alpha, or prostaglandin E2.
[0054]The potentiator may be a pharmacological agent or combination of agents that promote cellular processes that result in biological and/or mechanical creep and ultimately induce remodeling of the connective tissues that help define the size and shape of the penis. In addition, an agent which increases solubility of collagen may be used as a potentiator. Agents with very specific mechanisms of action may be used, or other agents with pleomorphic mechanisms of action, such as relaxin or growth hormone which trigger diverse mechanisms to induce growth in the penis may be used. For example, agents may be administered that facilitate the elongation of collagen fibers and accelerate the turnover remodeling rates of collagen through numerous mechanisms. For example, D-penicillamine and dimethyl sulfoxide (DMSO), which promote the elongation of collagen by inhibiting or interfering with inter- and intramolecular collagen cross-linkage may be used. Other agents include, but are not limited to, relaxin, insulin like growth factors, growth hormone, metalloproteinases or metalloproteinases agonists or promoters of collagenase activity, tissue inhibitors of matrix metalloprotenases (TIMPs) other agents that increase collagen solubility, prostaglandins, corticosteroids, or aminobenzoate potassium, a commercial brand being known as Potaba®. Preferred prostaglandins are prostaglandin F2 alpha and prostaglandin E2. Also included are pharmaceutically active sequences, peptidomimetics, or mimetics above the above-listed molecules.
Hey Mikeshlorts. With you saying to just use pge1 for 18 months keep in mind that I’ve been using pge1 now for over 5 months and have reached a point to here I need high dosages to have a good 2 to 4 hour erection if im not injecting close to bedtime. Right now at 90 mcgs it’s not getting me there and the pain is crazy with that much pge1. Not to mention at this point I can’t fit any more pge1 in a 1 CC syringe. I either need to find a source for a higher pge1 concentration or just use other stronger vasodilators to stack with a smaller amount of pge1 to reach 2 to 4 hour erections again like what was done in the patent and what Ronielle did after he reached 500 mcgs of pge1.
Keep in mind that yes there are a lot of theories going around but that’s the point of this thread. What Im doing is experimenting on myself to see what’s the optimal way of aheaving enlargement. So far I’ve gained a quarter inch in midgirth and base girth so this does work even for a vet like me. Right now I’m leaning towards the using what ever safe vasodilator necessary to achieve a nice tunica stretching 2 to 5 hour erection and starting to lean away from the goal of reaching 500 mcgs of PAINFUL pge1 like some of the test subjects did and Ronielle did.
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I'm talking to all your followers when I type posts criticizing the theorizers. I realize you've put in your time. I remind everyone that you got your length from 2+ years of hanging and manual Penis Enlargement.
Thanks for the info, great stuff.
Maybe you should just try titrating a 4 hour dose and take it before you go to bed. That's what Dr. Adams suggests.
Thanks for the info, great stuff.
Maybe you should just try titrating a 4 hour dose and take it before you go to bed. That's what Dr. Adams suggests.
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kingsnake;537598 said:
My subjective observation is, that most heavy gainers did something extreme/intense. Either time or force. sometimes both.
Erect jeqls. multiple sets of clamps all day. same with hanging. then short duration with much force. then the combination.
All of that will be multiplied by some chemicals. But is it the driving factor?
I know from myself, that too tough work will harden the tunica.. and its easier to create a hardened tunica then to soften a tunica(or even unlink)
The pain you feel..is it growing pain or toughening pain? Is the dmso+paba enough to prevent hardening of the tunica?
Cause we know from pe bro sience that someone, who hangs very heavy for a short time, will probably gain some, but will harden, when he hits maximum bearable force ..hindering all further gains..
So you might need some more time in between for low intensity/long time work.
Why not pge1 once a week and 2 days of vasilodators and dmso/paba following..Then extending or pumping.
Only read certain parts but very inspiring!
Any advice where I can research this? Any medical studys on these chemicals I can read about?
Thanks V.
Any advice where I can research this? Any medical studys on these chemicals I can read about?
Thanks V.
This study was done on fibroblast cells from the tunica of suffers of peyronies:
http://www.ncbi.nlm.nih.gov/puBathmateed/11002396
"Peyronie's disease is a fibromatosis of the tunica albuginea which affects up to 2% of men. Plaque development is believed to result, at least in part, from fibroblast proliferation and excess collagen deposition. Numerous oral and intralesional therapies have been used, including verapamil, colchicine and steroids. The purpose of this study was to investigate the in vitro effects of prostaglandin-E1 (PGE1), verapamil and colchicine on the proliferation rates of fibroblasts derived from Peyronie's disease tissue. Using tissue culture, multiple cell lines comprising fibroblasts from Peyronie's plaque, normal tunica and foreskin were established. Cells of low passage were removed from the parent culture and incubated with varying concentrations of PGE1 (0.1-10 mg/ml), verapamil (10-1000 mg/ml), and colchine (2.5 mg/ml). Proliferation was assessed at 48, 72 and 96 hours using the Vybrant MTT cell proliferation and then compared to control cells. Six plaque lines and 5 normal tunical cell lines were established. These cell lines exhibited excellent linear growth in culture media alone. Co-culture wih PGE1 resulted in no significant inhibition at 0.1 and 1 mg/ml, but a mean inhibition of 60.6+/-11.5% at a concenrtation of 10 mg/ml was noted. Similar inhibition was noted with verapamil at 100 and 1000 mg/ml with a mean inhibition of 65.2+/-10.6%. Colchicine resulted in a mean inhibition of 28% at a concentration of 2.5 mg/ml. Maximum inhibition occurred at 96 hours in all cases. There was no statisitically significant difference in proliferation rates between plaque and normal tunical cell lines. We have developed an in vitro model to assess the effects of biologically active agents on the growth of fibroblasts derived from Peyronie's disease tissue. Our data suggests that PGE1, verapamil, and colchicine inhibit in vitro proliferation of fibroblasts at specific concentrations. Refinement and application of this knowledge may allow the development of useful pharmacologic strategies for men with PD."
Therefore PGE1 may help reduce the production of collagen by fibroblasts in the tunica which would prevent toughening of the tunica. However, injecting into the cc doesn't get the PGE1 to where it is needed.
I have better things to do with my time than injecting my penis and doing Penis Enlargement enlargement exercises. I want a big dick as quickly as possible. Chemical Penis Enlargement shouldn't be this hard.
http://www.ncbi.nlm.nih.gov/puBathmateed/11002396
"Peyronie's disease is a fibromatosis of the tunica albuginea which affects up to 2% of men. Plaque development is believed to result, at least in part, from fibroblast proliferation and excess collagen deposition. Numerous oral and intralesional therapies have been used, including verapamil, colchicine and steroids. The purpose of this study was to investigate the in vitro effects of prostaglandin-E1 (PGE1), verapamil and colchicine on the proliferation rates of fibroblasts derived from Peyronie's disease tissue. Using tissue culture, multiple cell lines comprising fibroblasts from Peyronie's plaque, normal tunica and foreskin were established. Cells of low passage were removed from the parent culture and incubated with varying concentrations of PGE1 (0.1-10 mg/ml), verapamil (10-1000 mg/ml), and colchine (2.5 mg/ml). Proliferation was assessed at 48, 72 and 96 hours using the Vybrant MTT cell proliferation and then compared to control cells. Six plaque lines and 5 normal tunical cell lines were established. These cell lines exhibited excellent linear growth in culture media alone. Co-culture wih PGE1 resulted in no significant inhibition at 0.1 and 1 mg/ml, but a mean inhibition of 60.6+/-11.5% at a concenrtation of 10 mg/ml was noted. Similar inhibition was noted with verapamil at 100 and 1000 mg/ml with a mean inhibition of 65.2+/-10.6%. Colchicine resulted in a mean inhibition of 28% at a concentration of 2.5 mg/ml. Maximum inhibition occurred at 96 hours in all cases. There was no statisitically significant difference in proliferation rates between plaque and normal tunical cell lines. We have developed an in vitro model to assess the effects of biologically active agents on the growth of fibroblasts derived from Peyronie's disease tissue. Our data suggests that PGE1, verapamil, and colchicine inhibit in vitro proliferation of fibroblasts at specific concentrations. Refinement and application of this knowledge may allow the development of useful pharmacologic strategies for men with PD."
Therefore PGE1 may help reduce the production of collagen by fibroblasts in the tunica which would prevent toughening of the tunica. However, injecting into the cc doesn't get the PGE1 to where it is needed.
I have better things to do with my time than injecting my penis and doing Penis Enlargement enlargement exercises. I want a big dick as quickly as possible. Chemical Penis Enlargement shouldn't be this hard.
I say, once Snake perfects his pain-free chemical recipe, and we get the elongation benefits without all the crap he's had to go through, that we each give Snake the phone number of the first chick we get at post-enlargement!
Hey! This guy is being a guinea pig for all of us.
It's the least we can do...
Hey! This guy is being a guinea pig for all of us.
It's the least we can do...
bigdaddy777;538201 said:I say, once Snake perfects his pain-free chemical recipe, and we get the elongation benefits without all the crap he's had to go through, that we each give Snake the phone number of the first chick we get at post-enlargement!
Hey! This guy is being a guinea pig for all of us.
It's the least we can do...
It is important to give much respect to the trials and errors and eventual victories of KingSnakes persistence and tenacity! In all penis enlargement that treads on new territory there will always be a masters of these realms and this is where you find the innovation and advancements that are available.
Through my own research, both academic and self-observations, it seems that he is increasing his dosages of the drug far too often to achieve the desired end result. I've gone through many logs of others and have come to the conclusion that far too many users are titrating their dosages up too early and often. What I've seen is a negative return and a great cost to their bank account for no good reason. I've stayed on 6mcg- 8mcg for almost a month and I've not had any tolerance build-up or reduced return. In fact, I've gained in girth; sitting at 6in MEG.
I apologize if it has been posted recently but I may have missed it in this thread. Can someone post a link that KS is buying the DHT cream from?