FuzzyKen said:
Reference on Girth Enhancement Procedures.

The first thing to immediately rule out is any individual who has had alloderm or implantation of silicone these individuals are out of the question.

The next group were those who underwent either lipotransfer which was injection of fat and the next unsuccessful method which was dermal grafts.

In the case of those two methods the main problems would come from small left over nodules remaining behind. As the penis would begin to stretch the loose skin which you have left over would begin to tighten up again. (This is the good part) What could also happen however is that if there are nodules left behind, that something that was NOT a problem could now become evident as the lose skin tightens with the expansion of your penis. The loose skin hides a multitude of sins in many of these cases, so after the graft or transfer reabsorbed you may not see things that could be a problem. This is NOT a 100% thing, but what I would suggest is that the inital trial groups consist of individuals who are "virgin" in as many ways as is possible to all other methods of Penis Enlargement. It will give the data more weight, and then the others come into this later. As time goes on then bring in everything in a controlled manner. By doing this the experiences of the first group may be able to head off problems for later groups.

Fuzzy Ken


lol, not sure how many pe virgins there are reading this LMAO
 
I can't work out how to reply to each question or paragraph like you do Sikkdog :)

Regarding Potaba and p-Aminobenzoic Acid Potassium Salt, they do appear to be the same. I have emailed Potaba to ask if anything else is added.
If they are the same do you mind if I ask you why you would try to get Potaba from China if p-Aminobenzoic Acid Potassium Salt is readily available from research chemists at a seemingly low cost?

Sorry for taking the text out of context. I took it from this page where there is quite a bit of talk regarding the use of DHT cream. I just wondered what your opinion on this was because it appears some had concerns about using DHT cream with the Prostate being so near by, possible effects on the scalp hair. Also it's efficacy was questioned. Posts 470 to 479 were of interest.

http://www.mattersofsize.com/forum/showthread.php?t=5768&page=24&highlight=magnumforce
 
bluenun said:
I can't work out how to reply to each question or paragraph like you do Sikkdog :)
It's real easy... you just add "quote" (in braces) before the comment and "/quote" (in braces) after.

Regarding Potaba and p-Aminobenzoic Acid Potassium Salt, they do appear to be the same. I have emailed Potaba to ask if anything else is added.
If they are the same do you mind if I ask you why you would try to get Potaba from China if p-Aminobenzoic Acid Potassium Salt is readily available from research chemists at a seemingly low cost?
If it costs $5 a gram here in the US, it will cost $1 per gram in China... since i buy "other" powders from China now, it would just be more convenient to have a single source for everything.

Sorry for taking the text out of context. I took it from this page where there is quite a bit of talk regarding the use of DHT cream. I just wondered what your opinion on this was because it appears some had concerns about using DHT cream with the Prostate being so near by, possible effects on the scalp hair. Also it's efficacy was questioned. Posts 470 to 479 were of interest.

http://www.mattersofsize.com/forum/showthread.php?t=5768&page=24&highlight=magnumforce
I believe the concerns regarding DHT is overstated... at least for me it is. I don't suffer from MPB and it doesn't run in my family. I also know from first had experience that DHT does not affect my prostrate... i've been on heavy doese of test and other AAS with little effect on my prostrate. As i've mentioned on other threads, i routinely get bloodwork done when cycling and even when my DHT levels are elevated, i experience very little effects on my prostrate. Ephedra has had a more significant effect on my prostrate than any hormone that i've ever taken. Two day after taking an ephedra supplement, symptoms of enlarged prostrate manifests itself like: a strong urgency to urinate, having to urinate often, and difficulty when trying to urinate or empty bladder.

The discussions on DHT's efficacy was purely hypothetical/theroretical... according to the patent text, 3 out of 10 patients used DHT and 1 patient used testosterone exclusively as a potentiator with great results. I'll take anecdotal evidence over hypothetical blather any day of the week.

EXAMPLE 5

[0088] A male patient, age 44, was treated with intracavernosal injections of a quadruple mix of the vasodilators prostaglandin E1, Atropine, Chlorpromazine and Papavarine on a regular basis (approximately two to four times per week) over a 4-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement over a period of about 3 to 5 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator dihydrotestosterone 5% ointment was administered orally starting two weeks before starting the IC injections of the vasodilators Atropine, Chlorpromazine and Papavarine and prostaglandin.

[0089] After 4 months of treatment the patient's erect penis increased from 5.2 inches to 6.5 inches (about a 25% increase) in length.

EXAMPLE 6

[0090] A male patient, age 44, was treated with intracavernosal injections of the vasodilator phentolamine on a regular basis (approximately two to four times per week) over a 4-month treatment period. Phentolamine was frequently combined with indirect vasodilating effects of oral Viagra to produce and maintain a prolonged engorgement of 60-90% over a period of about 3 to 5 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator dihydrotestosterone gel was administered orally starting two weeks before starting the IC injections of the vasodilators Atropine, Chlorpromazine and Papavarine and prostaglandin.

[0091] After 4 months of treatment the patient's erect penis increased from 5.2 inches to 6.5 inches (about a 25% increase) in length.

EXAMPLE 8

[0093] A male patient, age 47, was treated with intracavernosal injections of a triple mix of the vasodilators Atropine, Chlorpromazine and Papavarine on a regular basis (approximately three to four times per week) over a 6-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement of an erectile response between 60-95% over a period of several hours, generally 3 to 4.5 hours. The quantity of medication was adjusted from time to time in accordance with the patient's response, which was monitored initially weekly. After 2 months of treatment subcutaneous injections of testosterone 14-20 mg into the penis were added as an accelerator.

[0094] The size of the patient's fully erect penis increased from 5.2 inches to 6.0 inches in length (about a 15% increase) over the 6 month treatment period.

EXAMPLE 10

[0096] A male patient, age 27, was treated with intracavernosal injections of a prostaglandin E1 on a regular basis (approximately two to five times per week) over a 3-month treatment period. Due to a sensitivity to Prostraglandin E1 causing aching and pain at higher doses, the maximum tolerated dose which produced a comfortable erection was only lasting 90 to 120 minutes. The patient used two separate IC injects spaced throughout the treatment days to achieve a total daily cumulative engorgement duration of 3 to 4 hours. The quantity of medication was adjusted in accordance with the patient's response. The 15 mg of the potentiator Dihydrotestosterone was injected subcutaenously into the penis daily throughout the treatment period. After 3 months of treatment the patient's erect penis increased from 6.3 inches to 7.1 inches (about an 13% increase) in length.
 
sikdogg said:
It's real easy... you just add "quote" (in braces) before the comment and "/quote" (in braces) after.

(quote)
If it costs $5 a gram here in the US, it will cost $1 per gram in China... since i buy "other" powders from China now, it would just be more convenient to have a single source for everything.(/quote)

Understood, makes good sense.

(quote)
I believe the concerns regarding DHT is overstated... at least for me it is. I don't suffer from MPB and it doesn't run in my family. I also know from first had experience that DHT does not affect my prostrate... i've been on heavy doese of test and other AAS with little effect on my prostrate. As i've mentioned on other threads, i routinely get bloodwork done when cycling and even when my DHT levels are elevated, i experience very little effects on my prostrate. Ephedra has had a more significant effect on my prostrate than any hormone that i've ever taken. Two day after taking an ephedra supplement, symptoms of enlarged prostrate manifests itself like: a strong urgency to urinate, having to urinate often, and difficulty when trying to urinate or empty bladder.(/quote)

Great to hear your experiences especially as you have had bloodwork whilst on test.

(quote)
The discussions on DHT's efficacy was purely hypothetical/theroretical... according to the patent text, 3 out of 10 patients used DHT and 1 patient used testosterone exclusively as a potentiator with great results. I'll take anecdotal evidence over hypothetical blather any day of the week.

Yes I noticed 3 of the 10 examples had used DHT. I assume where it says the 5% onitment and gel was administered orally it is a typo and means topically.
Example 5 and 6 look very familiar, age and both gained 25% but there are small differences in their treatment, strange.
 
Bro, braces are [] not ()...

bluenun said:
Yes I noticed 3 of the 10 examples had used DHT. I assume where it says the 5% onitment and gel was administered orally it is a typo and means topically.[/quote
I agree...

Example 5 and 6 look very familiar, age and both gained 25% but there are small differences in their treatment, strange.
It does look strange but the fact that gains were made with DHT and testosterone as potentiators is very promising.
 
reading the patent closely, the rate of gains seem to be determined of number of days that the vasodilators were used. personally im impressed with example 2
EXAMPLE 2

[0082] A male patient, age 30, was treated with intracavernosal injections of the vasodilator on a regular basis (approximately four to five times per week) over a 6-month treatment period. A sufficient quantity was administered to maintain a prolonged engorgement over a period of about 3 to 6 hours. The quantity of medication was adjusted in accordance with the patient's response. The potentiator potaba (aminobenzoate) (1000 mg/4 times per day) was administered orally to the patient for the last 60 days of treatment.

[0083] The patient's erect penis increased from 5.6 inches to 7.7 inches (about an 38% increase) in length and 3.2 inches to 5.3 inches (about an 65% increase) in girth over the 6-month treatment period.
here the potaba was only used during the last 2 months, although it doesnt state at which point most of the gains were made, is there any evidence at all that the DHT or potaba even aid the growth?. also the guy in example 1 seemed to be getting on well with the prostaglandin E1 on its own, hopefully this would be cheaper than buying 3 vasodilators as a trimix.
 
hey guys do you think i could simulate the vasodilatory injections by taking 50mg viagra and something like NO explode (body building supplement)?

Normally, sexual stimulation in the male is followed by release of nitric oxide (NO) in the corpus cavernosum of the penis. NO activates the enzyme guanylate cyclase in the smooth muscle cells which allows for the relaxation of the penile arteries and an increase flow of blood into the penile cavity. The effect of Viagra is to allow the accumulation of cGMP by inhibiting the phosphodiesterase action which tends to magnify the effect of NO release.

the plan is to take it just before bed along with the DHT cream and potaba. thats gotta keep me hard for a good few hours in the night, right? from my understanding of the patent its the engorgement that creates the gains, not necessarily how you got the engorgement. let me know what you think.
 
EVO said:
hey guys do you think i could simulate the vasodilatory injections by taking 50mg viagra and something like NO explode (body building supplement)?



the plan is to take it just before bed along with the DHT cream and potaba. thats gotta keep me hard for a good few hours in the night, right? from my understanding of the patent its the engorgement that creates the gains, not necessarily how you got the engorgement. let me know what you think.
I don't think that would keep you hard long enough... the problem with viagra/cialis is that they require stimulation to get and keep erections. Trimix and other injectable vasos don't...
 
sikdogg said:
Bro, braces are [] not ()...

bluenun said:
Yes I noticed 3 of the 10 examples had used DHT. I assume where it says the 5% onitment and gel was administered orally it is a typo and means topically.[/quote
I agree...

It does look strange but the fact that gains were made with DHT and testosterone as potentiators is very promising.

Yes it is.

Evo, I have been trying out cialis and 1 hour erect is about my limit before I get tired, it takes too much mental effort for me. That is why IC injections of Trimix or something similar is appealing for the goal of 3-6 hour erections even if they are at a lower level.
 
I received my Postaglandin a couple of days ago and I have to say that 5 micrograms can keep you hard for 3 hours.But not without any stimulation.
It´s just much easier to keep it up.Maybe a higher dosage would do the job,but I´m not planning to go up with the amount I inject.
Today I´m gonna add some potaba and I already inject 500mg of testosteron a week.Hope this will help me to gain another inch or two.
By the way the injections are not anything to be scarred about.And my dick just feels totally worn out after 3 hours with my cockring on.Sometimes I think that it could be a little too much.
 
Hey Large1,
First: what is your complete protocal & regimen for this.
Second: where did you purchase your prostaglandin from.


Many thanks.....
 
I get the prostaglandin from the pharmacy without prescription.Got my connections:)
Like I said before,I received it a few days ago.But I plan to inject 5 micrograms 4 days a week for at least 3 months,take 4 grams of potaba and shoot 500 mg of testosterone each week.Last but not least, I try to keep it erect for 3 hours (with a 1.25 inch rubber cockring/base girth is 6 inch).
With the 5 micrograms it´s not staying erect by itself but it´s not really difficult to keep it erect either.
Starting measurements are 8 inch bp x 5.5inch midshaft girth.
 
There are currently nine known receptors of prostaglandins on various cell types. Prostaglandins ligate a subfamily of cell surface seven transmembrane receptors G-protein-coupled receptors. These receptors are termed DP1-2, EP1-4, FP, IP, and TP, corresponding to the receptor that ligates the corresponding prostaglandin (e.g., DP1-2 receptors bind to PGD2). Prostaglandins thus act on a variety of cells such as vascular smooth muscle cells causing constriction or dilation, on platelets causing aggregation or disaggregation and on spinal neurons causing pain. Prostaglandins have a wide variety of actions, including, but not limited to muscular constriction and mediate inflammation. Other effects include calcium movement, hormone regulation and cell growth control. Thromboxane is created in platelets and causes vascular constriction and platelet aggregation. Prostacyclin comes from cells in the blood vessel walls and is antagonistic to thromboxane.

Prostaglandins are potent but have a short half-life before being inactivated and excreted. Therefore, they exert only a paracrine (locally active) or autocrine (acting on the same cell from which it is synthesized) function.

15-deoxy-Δ12,14-PGJ2 (15d-PGJ2) is a PGD2 derivative that acts on PPAR intracellular receptors.

This means prostaglandin WILL DIRECTLY effect the cardiac system (smooth muscle) increase contraction, therefore increase vasodilatation to counter the increase in cardiac output. This could trigger a myocardial infarction (heart attack) The heart and all muscles also rely on calcium to contract, this hormone increases calcium activity.
I'm not sure you realize what your fucking with.

I have an advance degree in Nursing, Although this may have some positive effects on the penis, IT WILL EFFECT OTHER BODY SYSTEMS.
 
The BTC Killer said:
There are currently nine known receptors of prostaglandins on various cell types. Prostaglandins ligate a subfamily of cell surface seven transmembrane receptors G-protein-coupled receptors. These receptors are termed DP1-2, EP1-4, FP, IP, and TP, corresponding to the receptor that ligates the corresponding prostaglandin (e.g., DP1-2 receptors bind to PGD2). Prostaglandins thus act on a variety of cells such as vascular smooth muscle cells causing constriction or dilation, on platelets causing aggregation or disaggregation and on spinal neurons causing pain. Prostaglandins have a wide variety of actions, including, but not limited to muscular constriction and mediate inflammation. Other effects include calcium movement, hormone regulation and cell growth control. Thromboxane is created in platelets and causes vascular constriction and platelet aggregation. Prostacyclin comes from cells in the blood vessel walls and is antagonistic to thromboxane.

Prostaglandins are potent but have a short half-life before being inactivated and excreted. Therefore, they exert only a paracrine (locally active) or autocrine (acting on the same cell from which it is synthesized) function.

15-deoxy-Δ12,14-PGJ2 (15d-PGJ2) is a PGD2 derivative that acts on PPAR intracellular receptors.

This means prostaglandin WILL DIRECTLY effect the cardiac system (smooth muscle) increase contraction, therefore increase vasodilatation to counter the increase in cardiac output. This could trigger a myocardial infarction (heart attack) The heart and all muscles also rely on calcium to contract, this hormone increases calcium activity.
I'm not sure you realize what your fucking with.

I have an advance degree in Nursing, Although this may have some positive effects on the penis, IT WILL EFFECT OTHER BODY SYSTEMS.
I hear what you're saying but most of the effects are localized due to the injection. The systemic affects should be small enough as to not casue any real issues. This is after all, a fairly routine medical treatment for ED. I speculate that the doses that are injected into the penis won't be any more risky that a good dose of viagra or cialis. Most ED medication we have today was originally designed for heart patients.

If it was possible to take these meds orally then your concern would be taken with more gravity. Since the effects would be systemic. In that sense... viagra/Cialis/Levitra pose a higher risk of myocardial infarction than a localized injection of prostaglandin.
 
I read in the patent that beta blockers could be used as the vasodialator.
I was on beta blockers for a while for some high blood pressure and it never gave me an erection. It seemed to make them harder to get. Do you think that is a hoax or is there a different way to take beta blockers to make you get an erection. I mean, putting it in the uretha or something. Any thoughts?
 
Another question. Are there no research companies that sell the prostoglandin?
 
chance said:
I read in the patent that beta blockers could be used as the vasodialator.
I was on beta blockers for a while for some high blood pressure and it never gave me an erection. It seemed to make them harder to get. Do you think that is a hoax or is there a different way to take beta blockers to make you get an erection. I mean, putting it in the uretha or something. Any thoughts?
The patent doc lists all sorts of compounds that may be used. I believe that this was done to cover as much area as possible. Doing this makes it difficult to copy their patent.

That said, not everyone reacts to every med the same. This is why there are many different types of beta blockers.
 
chance said:
Another question. Are there no research companies that sell the prostoglandin?
I know of at least one that sells PGE1 for $125 per 10ml vial (10mcg/ml) or $90 for 2 or more.
 
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