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Medically documented picture of a man...obviously black who got megallophallus as a side effect of sickle cell anemia. Just thought it would be interesting to share.
 
I speculated a while back in D0zer's thread on clamping (corpse dick thread) that the sickle cell gene is atleast partly responsible for the larger penises often seen in black men. This certainly helps add credibility to my idea. Thanks SWM!
 
Men with sickle cell have a high incidence of repeated priapism. I believe that is the cause of the megalophallus, and not the genes.
 
Vaseline_Knight said:
how do u know if u have sickle-cell gene? and is it a good thing?

this is how you know

Sickle cell anemia is the most common inherited blood disorder in the United States, affecting about 72,000 Americans or 1 in 500 African Americans. SCA is characterized by episodes of pain, chronic hemolytic anemia and severe infections, usually beginning in early childhood.


Doesnt sound good to me :s

http://www.ncbi.nlm.nih.gov/books/bv.fcgi?call=bv.View..ShowSection&rid=gnd.section.98
 
Vaseline_Knight said:
ok wtf? even when they get a bad disease there's a lil gift in it for them. i dont see my dick grow when I get sick <:(

With gene therapy being advanced as it is...if u have enough money a crazy enough doctor could put the SCA in u dunno if itll work tho...maybe it will make u sicker without a bigger penis so u spend thousands just to get sicker MUWHAHAHAAHHAHAHAHAHA rofl
 
I speculated a while back in D0zer's thread on clamping (corpse dick thread) that the sickle cell gene is atleast partly responsible for the larger penises often seen in black men. This certainly helps add credibility to my idea. Thanks SWM!

Interesting.
 
Complications
Sickle cell anemia can lead to a host of complications, including:

Stroke. A stroke can occur if sickle cells block blood flow to an area of your brain. Stroke is one of the most serious complications of the disease. Signs of stroke include seizures, weakness or numbness of your arms and legs, sudden speech difficulties, and loss of consciousness. If your baby or child has any of these signs and symptoms, seek medical treatment immediately. A stroke can be fatal.

Acute chest syndrome. This life-threatening complication of sickle cell anemia causes chest pain, fever and difficulty breathing. Acute chest syndrome is similar to pneumonia, but is caused by a lung infection or trapped sickle cells in your lungs. It requires emergency medical treatment with antibiotics, blood transfusions and drugs that open up airways in your lungs. Recurrent attacks can damage your lungs.

Organ damage. Sickle cells can block blood flow through blood vessels, immediately depriving an organ of blood and oxygen. In sickle cell anemia, blood is also chronically low on oxygen. Chronic deprivation of oxygen-rich blood can damage nerves and organs in your body, including your kidneys, liver and spleen. Organ damage can be fatal.

Blindness. Tiny blood vessels that feed your eyes can get plugged with sickle cells. Over time, this can damage the retina — the portion of each eye that processes visual images — and lead to blindness.

Other complications. Sickle cell anemia can cause open sores, called ulcers, on your legs. Sickle cells can block blood vessels that nourish your skin, causing skin cells to die. Once skin is damaged, sores can develop. Gallstones also are a possible complication. The breakdown of red blood cells produces a substance called bilirubin. Bilirubin is responsible for yellowing of the skin and eyes (jaundice) in people with sickle cell anemia. A high level of bilirubin in your body can also lead to gallstones. Men with sickle cell anemia may experience painful erections, a condition called priapism. Sickle cells can prevent blood flow out of an erect penis. Over time, priapism can damage the penis and lead to impotence in men with sickle cell anemia.

SOURCE:http://www.mayoclinic.com/health/sickle-cell-anemia/DS00324/DSECTION=6


Still think SCA is sexy??? rofl
 
Interesting for sure. I wonder how many cases of priapism and the duration of erection resulted in permanent enlargement.

If the priapism is caused by abnormal blood cells somehow blocking the outflowing of blood from the penis, resulting in a greater volume of blood entering the penis than leaving, result being permanent stretching and enlargement. Is lymph flow affected?? How much oxygen stays with the hemoglobin as compared to pumping or clamping, during priapism??

There must have been a post healing/recovery phase after the priapism.
 
It is somehow not mentioned that ONLY AFRICAN AMERICANS have this sickle cell gene. Many carry it, but very few in comparison HAVE the disease becaus eit is a recessive trait. Ironically, inheriting the gene from one parent but not the other (multi-racial or comletely black) is advantageous, because it protects against malaria, but is not enough to cause the disease. It does have some affects like astHydromaxa. It is possible that carrying the gene (but not having the disease) may be enough to cause mini cases of priapism, but not serious ones, which would mimic a constrictor session, and have the added effect of cutting oxygen. It would be interesting to hear from someone who has this disease or carries the recessive gene.

Also, NO DOCTOR WILL EVER INSERT THUS GENE INTO ONE's DNA!!!
 
What got me about these two photos is what the medical profession identifies as "megalophallus". Somehow, I expected even BIGGER !

(I've been around penis websites too long and my judgment is skewed.:s )

I dunno. When I have heard the term "mega" , I've associated it with really super-DOOPenis EnlargementR large sizes and granted, what's shown here is much larger than average, to be sure. I also found it intersting a flaccid size was used in these medical photos (calling THEM "mega") when a large erect size wasn't necessarily being depicted. Goodness knows what the docs would call some of what's on the internet (the ones which aren't photo-shopped of course!)

With our access to thousands of penises now (pix that is), I'm afraid many of us are fast loosing all objectivity on the subject of the phallus! ?:(
 
benderten2001 said:
What got me about these two photos is what the medical profession identifies as "megalophallus". Somehow, I expected even BIGGER !

(I've been around penis websites too long and my judgment is skewed.:s )

I dunno. When I have heard the term "mega" , I've associated it with really super-DOOPenis EnlargementR large sizes and granted, what's shown here is much larger than average, to be sure. I also found it intersting a flaccid size was used in these medical photos (calling THEM "mega") when a large erect size wasn't necessarily being depicted. Goodness knows what the docs would call some of what's on the internet (the ones which aren't photo-shopped of course!)

With our access to thousands of penises now (pix that is), I'm afraid many of us are fast loosing all objectivity on the subject of the phallus! ?:(

Hit the nail on the head
 
The exact cause of megalophallus is unknown, but it is most common in men with sickle cell disease. There have been several theories regarding the disorder. A popular idea is that the prolonged entrapment of blood blocks new oxygen from entering the penis, which effectively causes hypertrophy (growth). Another idea suggests that the prolonged erection stretches some of the tissues within the penis, along with exercising the penile smooth muscle.

Nevertheless, before you consider a way of giving yourself a priapism, know that a large percentage of the men who experience them become impotent.

Sources:

Datta, N. S. Megalophallus in sickle cell disease .&rdquo Journal of Urology. May 1977, pp. 672-673.

Kassim, A. A., et al. Megalophallus as a Sequela of Priapism in Sickle Cell Anemia : Use of Blood Oxygen Level-Dependent Magnetic Resonance Imaging.&rdquo Urology. Vol. 56 (2000), No. 3, pp. 509.
 
"Megalophallus as a sequela of priapism in sickle cell anemia: use of blood oxygen level-dependent magnetic resonance imaging.

Kassim AA, Umans H, Nagel RL, Fabry ME.

Department of Medicine (Division of Hematology), Albert Einstein College of Medicine, Montefiore Medical Center, Bronx, New York 10461, USA.

Priapism is a common complication of sickle cell anemia. We report a little known sequela of priapism: painless megalophallus, with significant penile enlargement. The patient had had an intense episode of priapism 9 years previously and his penis remained enlarged. Blood oxygen level-dependent magnetic resonance imaging revealed enlarged, hypoxic corpora cavernosa. Megalophallus probably resulted from permanent loss of elasticity of the tunica albuginea due to severe engorgement during the episode of priapism. This sequela needs to be recognized by physicians because no intervention is necessary and sexual function seems to remain intact."
 
1: J Natl Med Assoc. 1993 Jun;85(6):473-4. Related Articles, Links


An unusual case of priapism.

Jam M, Datta NS, Askari A.

Department of Surgery, King-Drew Medical Center, Charles R. Drew University of Medicine and Science, Los Angeles, CA 90059.

A case of sickle cell disease with 63 documented episodes of priapism that were managed medically is presented. The case is very unusual because of the fact that despite so many episodes of priapism, he did not lose sexual potency. On the contrary, over a period of time, his penis hypertrophied. To the best of our knowledge, this is the first such case with so many episodes of priapism reported in the English literature. We present a hypothesis for such unusual occurrence.

PMID: 8366540 [PubMed - indexed for MEDLINE]
 
Reduction Phalloplasty and Penile Prosthesis Implantation in a Case of Megalophallus as a Consequence of Neglected Priapism
Kattan, S 1 ; Seyam , RM 1 ; Al-Ghamdi, D 1
1 : King Faisal Specialist Hospital and Research Center
Copyright Journal of Sexual Medicine 1743 6095
ABSTRACT

Objectives: Megalophallus is a rare complication of priapism and sickle cell disease. More commonly, priapism in those patients is recurrent, difficult to prevent and ends up in erectile dysfunction as a result of cavernous tissue fibrosis or surgical intervention. A 33 year old man presented to us with a history of neglected priapism for 12 days 2 years previously. He developed a permanent unusual enlargement of the penis and erectile dysfunction. The patient determined that his new penile size is nonfunctional and unacceptable. We evaluated the patient and set out to surgically fashion the penis into a usable size, with adequate rigidity and stable condition.

Material and Methods: For 9 years the patient had frequent episodes of priapism which resolved spontaneously. The last episode was unsuccessfully treated conservatively and eventually the patient received a Winter shunt. The penis became enlarged and erections soft. Upon examination with intracavernous injection of 60 mg papaverine, the penis became mildly engorged and it measured 21 cm in circumference and 18 cm in length from the pubic pad of fat to the tip of the glans. MRI showed that the enlargement is mainly due to massive enlargement of corpora cavernosa. We based our goals on normative data for penile length and circumference. Under general anesthesia, we degloved the penis using a circumcision incision. The tunica albuginea was exposed laterally. A 3 cm wide strip was marked on the lateral side of each corpus cavernosum and excised along the long axis of the penis from 1 cm proximal to the coronal sulcus to the pubic bone. The cavernous tissue underneath was fibrotic and was excised to fit in the cylinders of an AMS 700 Ultrex penile prothesis. The tunica albuginea was run with an absorbable suture and the pump and reservoir were placed in the scrotum and retropubic space. Inflation of the prosthesis resulted in rupture of the suture line for 5 cm on the right side just proximal to the glans. Repeated removal of underlying cavernous tissue was not enough to accommodate the cylinder and a free tunica albuginea patch graft was sewn in.

Results: At the end of surgery, the penile length was left intact, while the circumference was reduced to 15 cm.

Conclusion: Reduction phalloplasty is feasible to manage the unusual cases of megalophallus. Extensive dissection and cavernous tissue removal necessitate penile prosthesis implantation. This is particularly justified in cases associated with erectile dysfunction complicating priapism.
 
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