Less oxygen = more growth
Still at it! Wasn't going to report back until I had a significant story to tell lol. I hate when people report 0.1 or 0.2 inch gains that could just be EQ. I was going to wait until I have atleast a half inch to tell about. My eq hasn't been great because I've been having A LOT of sex. I really need to chill out with that I know. But I've still been clamping for atleast an hour straight per day and making sure my penis turns Dark purple. I've noticed that when it gets very dark colored, white spots start to appear and that freaks me out so I un-clamp at that point. No huge noticeable gains as of now. I was gonna go another couple weeks and then measure and see what's going on. Will definitely let you guys know when I have a legit solid gain measurement to give ya. I promise
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Xxxi;727431 said:Still at it! Wasn't going to report back until I had a significant story to tell lol. I hate when people report 0.1 or 0.2 inch gains that could just be EQ. I was going to wait until I have atleast a half inch to tell about. My eq hasn't been great because I've been having A LOT of sex. I really need to chill out with that I know. But I've still been clamping for atleast an hour straight per day and making sure my penis turns Dark purple. I've noticed that when it gets very dark colored, white spots start to appear and that freaks me out so I un-clamp at that point. No huge noticeable gains as of now. I was gonna go another couple weeks and then measure and see what's going on. Will definitely let you guys know when I have a legit solid gain measurement to give ya. I promise
Good Shit! Thanks for pushing onward!
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If this doesn't pan out, you might try approaching this like weight hanging. In other words, multiple 15-20 minute sets of high pressure clamping. Do a set, break for a few minutes with a return to full flaccid, repeat.
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This is always such an interesting topic. Im pretty sure that the expansion is the #1 factor in size increase even with pripism. Cause many big gainers have achieved gains through that either by intensity or time or both under expansion. Not many report of going towards numbness. So clearly there is no definite need to achieve that low oxygen enviroment to gain.
Most megalophallus seem to happen through low flow priapism where the dick basicly baloons up creating more and more expansion without blood leaving. The other is high flow priapism.
Im clamping for many years and after some minutes totally clamped off it gets very dark and cold already.
I cant imagine having stale blood for 1.5 hours without any fresh one in.
I could clamp for a long time kegeling in fresh blood all the time and let some of the old leave.
What does the temperature feel like? It must feel very cold like ambient room temperature at the most?
I cant imagine you having not tissue death after 20-40 minutes.
Which size do you reach while clamping? Bigger then your normal erect size? Or are you not aiming for expansion but simply for oxygen deprivation?
Most megalophallus seem to happen through low flow priapism where the dick basicly baloons up creating more and more expansion without blood leaving. The other is high flow priapism.
Xxxi;726825 said:
Im clamping for many years and after some minutes totally clamped off it gets very dark and cold already.
I cant imagine having stale blood for 1.5 hours without any fresh one in.
I could clamp for a long time kegeling in fresh blood all the time and let some of the old leave.
What does the temperature feel like? It must feel very cold like ambient room temperature at the most?
I cant imagine you having not tissue death after 20-40 minutes.
So you clamp of a semi erection?Xxxi;726861 said:
Which size do you reach while clamping? Bigger then your normal erect size? Or are you not aiming for expansion but simply for oxygen deprivation?
I can't get a 100% rock hard erection clamped off. Never have been able to. It always ends up somewhat squishy lol. So I clamp off an 80-90% erection. Main goal is less oxygen.
I don't think priapism megalophallus is caused by the pressure. I'll explain why. In every medical case I've seen of megalophallus after priapism, the CC was reported hypoxic. I also believe megalophallus has tissue growth, not just a stretched out tunica. If it were just a crazy tunica stretch, then the penis would actually shrink when flaccid. However, megalophallus penises stay the same size when flaccid and just get harder when erect. That is because there is new tissue in there. Also, to actually stretch out the tunica THAT much would take MASSIVE pressure. We all know how tough the tunica is. And to get enough pressure to deform it to the extent of a 8 or 10in girth (megalophallus size), you'd basically need arteries that pump blood into it with the force of a damn pressure washer imo. I really don't think you can just "engorge" to the point of deforming the tunica that much.
In one case I read, it says the megalophallus was caused by a "sudden loss of tunica elasticity". I believe that was because of a chemical change within the structure of the tunica (probably caused by lack of oxygen). And in another case, when a megalophallus REDUCTION surgery was done, the doctors reported cutting out and removing bleeding TISSUE. So, I think some kind of chemical reaction is what is causing the tunica to weaken soo much and new tissue to actually grow. There's no way this (kind of megalophallus size gain) is just caused by pressure and sheer force.
I don't think priapism megalophallus is caused by the pressure. I'll explain why. In every medical case I've seen of megalophallus after priapism, the CC was reported hypoxic. I also believe megalophallus has tissue growth, not just a stretched out tunica. If it were just a crazy tunica stretch, then the penis would actually shrink when flaccid. However, megalophallus penises stay the same size when flaccid and just get harder when erect. That is because there is new tissue in there. Also, to actually stretch out the tunica THAT much would take MASSIVE pressure. We all know how tough the tunica is. And to get enough pressure to deform it to the extent of a 8 or 10in girth (megalophallus size), you'd basically need arteries that pump blood into it with the force of a damn pressure washer imo. I really don't think you can just "engorge" to the point of deforming the tunica that much.
In one case I read, it says the megalophallus was caused by a "sudden loss of tunica elasticity". I believe that was because of a chemical change within the structure of the tunica (probably caused by lack of oxygen). And in another case, when a megalophallus REDUCTION surgery was done, the doctors reported cutting out and removing bleeding TISSUE. So, I think some kind of chemical reaction is what is causing the tunica to weaken soo much and new tissue to actually grow. There's no way this (kind of megalophallus size gain) is just caused by pressure and sheer force.
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Well you are trying to create a unique medical occurrence (that doesn't always end well). Using proper techniques like stretching and water pumping you maybe, MAYBE, will get ur dream size. Not trying to poo poo you just trying to help out. Also IMO rock hard bullet proof EQ would be the first step of creating a beneficial priapism. You are just cruising for a bruising, literally.
Xxxi;727598 said:
I'm glad that you specified "less oxygen" because its important to know its not the same as "no oxygen" = anoxia (just a little background for those that may not know; not something that I think you'd want to play with to be honest).
I can't say for sure that anything mentioned here would be fact but I'm only writing from what I remember in readings. The tunica has what's like a pressure point for elasticity. When it reaches this point it sort of "gives up". This is why megalophallus caused by priapism doesn't shrink when flaccid, that's not to say there aren't any tissue growth. I'd like to look at the medical cases you are citing for growth if you get the chance, you can just pm them to me. The few cases I've seen regarding this, patient testimonies say its almost an instant increase in girth, something that would be impossible by growth due to the rate at which cell division would need to occur in that short amount of time. That's not to say growth doesn't occur later to stabilize this change, I do not know.
About the reduction surgery, I went thru the case study for it shortly after we first started discussing this. The bleeding tissue was mostly corpus cavernosum so its already very vascular, so I'm not surprised it was capable of bleeding.
To be fair, some support on the other side of the coin, pressure, can be made plausible. Let's look at a normal erection. The spongy tissue fills with blood, from arteries down the length of the penis. A little blood enters the corpus spongiosum; the remainder engorges the corpora cavernosa (about 90% of the blood used in an erection). Blood can leave the erectile tissue only through a drainage system of veins around the outside wall of the corpus cavernosum (in the case of priapism, there is usually something causing an inability to drain properly). The expanding spongy tissue presses against a surrounding dense tissue (tunica albuginea) constricting these veins, preventing blood from leaving (essentially to hits on no drainage). The penis becomes rigid as a result. The glans penis, the expanded cap of the corpus spongiosum, remains more malleable during erection because its tunica albuginea is much thinner than elsewhere in the penis. That being said, its not impossible with blood being blocked inside the penis by two different mechanisms (while inflow is not impeded) to have something that will yield to the increasing pressure. If something didn't yield, the penis would pop like an over-inflated balloon. I think that would explain a sudden loss in tunica elasticity.
Your experiment may provide some further insight into hypoxic growth, only wanted to highlight that it would not be out of the realm of possibility for this to be a pressure thing. Look at the cases of fractured penises, that's usually caused by a ruptured tunica so its not impervious to pressure deformation. Not many ways to find out for sure the causes, could even be a combination of hypoxic conditions and excessive pressure.
Keep up with your experiment and share your findings! Don't be afraid to discontinue if something goes wrong, protect yourself first.
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I was thinking on this and when I was doing this experiment, in my own way, I was keeping an erection for at the minimum 2 hours, I would try to hold it for 4. I do not think one hour would cause the mega-phallus that would occur due to priapism.
doublelongdaddy;727624 said:
I think this may be a bit of anecdotal support towards a combination of both. Prolonged hypoxic conditions (reduced blood outflow to maintain erection but with some oxgen inflow) and extended episode of pressure (exerted from maintaining an erection).
All good points here. The thing about the penis reduction surgery megalophallus case that gets me is that it said the growth resulted after 3 separate priapisms. That's what led me to believe that it was a result of tissue growth. I've had ROCK hard PGE 1 erections before and no size gain immediately as in "tunica giving up". I really don't see how someone could get enough pressure to stretch the tunica just via pressure. I wish I did know tho, I'd do it tonite LOL
Xxxi;727665 said:
Well see the main difference between your induced erection and priapism is that your "plumbing" still works. Your body won't allow damage to be done by pressure because your defense mechanisms are still functional. With priapism, something is mechanically blocking the path (sickled RBCs in the case of sickle cell anemia).
I'm gonna try to explain that concept in a way that makes it simpler for us. Let's say you have a water hose, you kink the hose and it slows down weather out flow and the pressure inside the tube is increased. This is a normal erection (doesn't matter if it's chemical or natural). If the pressure gets higher and higher the kink will loosen a bit to let more water out because it is the weak point. Now let's take something and block that hose internally. Let's take a ball (representing the clump sickled cells can form) that can stretch the malleable part of the hose but can't get past the thread connection (equivalent to venous valves in this example). No matter how much the pressure increases behind the ball, it won't let more water out than possible because it's clogged at the threaded connection (the strong point of the hose). It leaves only the weaker components of the hose to relieve that pressure. This would be like priapism. Did that make sense or did I fudge it up even more? Lol
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chargains;727668 said:
I do understand what you are saying. But back to the hose example, even if you totally kink it or put a stopper in it, it's not going to build up enough pressure to stretch the hose rubber (tunica). It's just going to be stagnant at the hoses expansion limit because the hose rubber is too stiff to expand just due to the pressure of the water. You'd need more water pressure to expand the hose beyond its pre-made diameter.
I think that is how the penis is. Even if you block off the outflow completely, I really don't see how you could get enough IN-FLOW PRESSURE to keep forcing more blood in. The tunica is soo tough and rigid, to have enough pressure to stretch it would have to be incredible pressure and I don't think the arteries are capable of pushing blood with that much force. I think the tunica has to be weakened by some other means first before mere blood pressure can expand it beyond its genetic limit.
I hope that made sense.
I mean, even with ssj, smashes, all kinds of intense clamping and compression, we still aren't able to force the tunica to "give up" and expand to massive sizes like some of these priapisms do. Do you think that the pressure from a heart pushing blood through an artery is really exerting more force than DLD when he's squashing his penis with the brute force of his hands?
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Xxxi;727684 said:
I agree. I only said that to illustrate the difference between a PGE-1 erection vs one caused by priapism. That wasn't speaking specifically about the deformation you're seeking.
Thats why I think it may be a combination effort that's causing the expansion. I don't think it is necessarily something to be attributed to one factor regardless of how significant each individual factor may seem. It's more like a cascade of events and conditions that have that outcome.
A little random information tho, the tensile strength of the average tunica is roughly 1500 mmHg. The heart's pressure in a normal person (not pressure measured peripherally in the arms or legs) is 10^4 Pascal ~ 750mmHg. Reaching tensile strength would rupture the tunica but the heart itself could get enough pressure for deformation if it wasn't impeded. Loss of elasticity would mean a sudden stretch from that build up. I don't think the force from hand work is less than what the heart is capable of but one is on a macro level and the other a micro level so pressure difference would be very different. [Also a difference in this example would be between water and blood. Blood has formed components that exert pressure in a different way from the molecules of water. Its part of the reason high blood pressure is damaging to microstructures.]
But yes, I can understand how you don't think it's just pressure, neither do I. I'm only saying that pressure can be a compounding factor in addition to whatever else is causing the change. Hopefully you can figure that out with your experiment.
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Thanks for the feedback Chargains. I really appreciate all your input and support. I think me and you both have realized that this could be the end-all of PE if we can finally nail it. I know some people are very against this kind of risky experimenting, but the thing that drives me is how CLOSE I feel like we are. We know it's between pressure and hypoxia. Those are the keys. If we can just figure out the correct way to apply those two factors, PE will never be the same again.
Haha and then if we could link up with DLD to make a user-friendly pump/clamp device that could keep the penis engorged while cutting off the oxygen in the right way to replicate megalophallus, boom, were multi millionaires! lol
Haha and then if we could link up with DLD to make a user-friendly pump/clamp device that could keep the penis engorged while cutting off the oxygen in the right way to replicate megalophallus, boom, were multi millionaires!
lolXxxi;727710 said:Thanks for the feedback Chargains. I really appreciate all your input and support. I think me and you both have realized that this could be the end-all of PE if we can finally nail it. I know some people are very against this kind of risky experimenting, but the thing that drives me is how CLOSE I feel like we are. We know it's between pressure and hypoxia. Those are the keys. If we can just figure out the correct way to apply those two factors, PE will never be the same again.
Haha and then if we could link up with DLD to make a user-friendly pump/clamp device that could keep the penis engorged while cutting off the oxygen in the right way to replicate megalophallus, boom, were multi millionaires!
I really want you to find some success with this. Guys have been trying for a while without real notable progress with this type of growth. Its going to take some work to isolate the causes and replicate the conditions with minimum damage. You definitely sound like you don't mind putting in the work and that is very admirable. I don't have as much time as I would like to help research this but when I do, I will post it here as usual. I think the more that's added to the knowledge pool regarding this, the better we'll be able to target what the change would be.
That's why your experiment should provide some valuable insight into the hypoxic aspect and the growth associated with that. If you're successful, even marginally so, we as community could have better insight as to were to go from there. Like I keep saying lol, keep it up and bring your ideas, one day we'll find out what makes this work.
