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what is everyones opinion on them? should only people who are athletes, body builders, etc use them? are they worth the risk?
EVO said:i have been on a few steroids myself:- testex, decanoate, oxy bolone, napasims, provirones, hcg, testivirone. my advice is this:- know what you are doing! many of my friends still do them and moan to me that their balls are shrinking. but they cant come off steroids because then their muscle mass will suffer. its a lose - lose situation. i recommended that my friend do the following cycle though - 2 months of decanoate and testivirone, one month on just hcg. then repeat. - take the next 6 months off. this is a years routine! the hcg is there to prevent your balls giving in! i do not recomend the use of steroids as the results are only temporary (some would disagee...) but if you do - i suggest a cycle as above...
EVO said:what does pct mean? ?![]()
WRONG... HCG helps to kickstarts the testes which will help with recovery but you don't seem to understand what's really going on. When you increase test levels exogenously, estrogen levels will increase also to maintain the test/estro ratio... when you come off steroids, test levels drop off pretty quick because your body is no longer producing it but estro levels are still high. This is when you're most prone to estrogen sides. It is imperative that you take a good anti-estrogen to block it's effects 'til your natty test levels come back. Clomid/Nolva are estrogn blockers that will block the effects of estro and also encourage your HTPA to begin working again. You also don't want to take HCG post cycle because it is also suppressive and will inhibit recovery.EVO said:rofl think thats basically what the hcg does....
That article is good but the HCG part is a little dated... lower doses run throughout the cycle works better.prince Albert said:Lol thats the reason your gains are only temperary mate
PCT is post cycle therapy.
little article that will give you a basic idea
http://www.muscletalk.co.uk/clomid-hcg.asp
http://www.ncbi.nlm.nih.gov/entrez/...f&pmid=15713727
LOW DOSE HUMAN CHORIONIC GONADOTROPIN MAINTAINS INTRATESTICULAR TESTOSTERONE IN NORMAL MEN WITH TESTOSTERONE INDUCED GONADOTROPIN SUPPRESSION.
Coviello AD, Matsumoto AM, Bremner WJ, Herbst KL, Amory JK, Anawalt BD, Sutton PR, Wright WW, Brown TR, Yan X, Zirkin BR, Jarow JP.
Center for Research in Reproduction and Contraception, Geriatric Research Education and Clinical Center, Veteran Affairs Puget Sound Health Care System (AMM), and Department of Medicine, University of Washington School of Medicine (ADC, WJB, JKA, BDA, PLS), Seattle, WA; Department of Medicine, Charles R. Drew University (KLH), Los Angeles, CA; Department of Urology, Johns Hopkins University School of Medicine (XY, JPJ), Baltimore, MD; Division of Reproductive Biology, Department of Biochemistry and Molecular Biology Johns Hopkins University School of Public Health (WWW, TRB, XY, BRZ, JPJ), Baltimore, MD.
In previous studies of testicular biopsy tissue from healthy men, intratesticular testosterone (ITT) has been shown to be much higher than serum testosterone (T), suggesting that high ITT is needed relative to serum T for normal spermatogenesis in men. However, the quantitative relationship between ITT and spermatogenesis is not known. To begin to address this issue experimentally we sought to determine the dose response relationship between human chorionic gonadotropin (hCG) and ITT to determine the minimum dose needed to maintain ITT in the normal range. Twenty-nine men with normal reproductive physiology were randomized to receive 200 mg T enanthate (TE) weekly in combination with either saline placebo or hCG 125 IU, 250 IU, or 500 IU every other day for 3 weeks. ITT was assessed in testicular fluid obtained by percutaneous fine needle aspiration at baseline and the end of treatment. Baseline serum T (14.1 nmol/L) was 1.2% of ITT (1174 nmol/L). LH and FSH were profoundly suppressed to 5% and 3% of baseline respectively, and ITT was suppressed by 94% (1234 nmol/L to 72 nmol/L) in the TE/placebo group. ITT increased linearly with increasing hCG dose (P < 0.001). Post-treatment ITT was 25% less than baseline in the 125 IU hCG group, 7% less than baseline in the 250 IU hCG group, and 26% greater than baseline in the 500 IU hCG group. These results demonstrate that relatively low dose hCG maintains ITT within the normal range in healthy men with gonadotropin suppression. Extensions of this study will allow determination of the ITT concentration threshold required to maintain spermatogenesis in man.
sikdogg said:You also don't want to take HCG post cycle because it is also suppressive and will inhibit recovery.
EVO said:what does it suppress sikdogg? can you post this info, cheers