Xxxi;727598 said:
I can't get a 100% rock hard erection clamped off. Never have been able to. It always ends up somewhat squishy lol. So I clamp off an 80-90% erection. Main goal is less oxygen.

I don't think priapism megalophallus is caused by the pressure. I'll explain why. In every medical case I've seen of megalophallus after priapism, the CC was reported hypoxic. I also believe megalophallus has tissue growth, not just a stretched out tunica. If it were just a crazy tunica stretch, then the penis would actually shrink when flaccid. However, megalophallus penises stay the same size when flaccid and just get harder when erect. That is because there is new tissue in there. Also, to actually stretch out the tunica THAT much would take MASSIVE pressure. We all know how tough the tunica is. And to get enough pressure to deform it to the extent of a 8 or 10in girth (megalophallus size), you'd basically need arteries that pump blood into it with the force of a damn pressure washer imo. I really don't think you can just "engorge" to the point of deforming the tunica that much.

In one case I read, it says the megalophallus was caused by a "sudden loss of tunica elasticity". I believe that was because of a chemical change within the structure of the tunica (probably caused by lack of oxygen). And in another case, when a megalophallus REDUCTION surgery was done, the doctors reported cutting out and removing bleeding TISSUE. So, I think some kind of chemical reaction is what is causing the tunica to weaken soo much and new tissue to actually grow. There's no way this (kind of megalophallus size gain) is just caused by pressure and sheer force.

I'm glad that you specified "less oxygen" because its important to know its not the same as "no oxygen" = anoxia (just a little background for those that may not know; not something that I think you'd want to play with to be honest).

I can't say for sure that anything mentioned here would be fact but I'm only writing from what I remember in readings. The tunica has what's like a pressure point for elasticity. When it reaches this point it sort of "gives up". This is why megalophallus caused by priapism doesn't shrink when flaccid, that's not to say there aren't any tissue growth. I'd like to look at the medical cases you are citing for growth if you get the chance, you can just pm them to me. The few cases I've seen regarding this, patient testimonies say its almost an instant increase in girth, something that would be impossible by growth due to the rate at which cell division would need to occur in that short amount of time. That's not to say growth doesn't occur later to stabilize this change, I do not know.

About the reduction surgery, I went thru the case study for it shortly after we first started discussing this. The bleeding tissue was mostly corpus cavernosum so its already very vascular, so I'm not surprised it was capable of bleeding.

To be fair, some support on the other side of the coin, pressure, can be made plausible. Let's look at a normal erection. The spongy tissue fills with blood, from arteries down the length of the penis. A little blood enters the corpus spongiosum; the remainder engorges the corpora cavernosa (about 90% of the blood used in an erection). Blood can leave the erectile tissue only through a drainage system of veins around the outside wall of the corpus cavernosum (in the case of priapism, there is usually something causing an inability to drain properly). The expanding spongy tissue presses against a surrounding dense tissue (tunica albuginea) constricting these veins, preventing blood from leaving (essentially to hits on no drainage). The penis becomes rigid as a result. The glans penis, the expanded cap of the corpus spongiosum, remains more malleable during erection because its tunica albuginea is much thinner than elsewhere in the penis. That being said, its not impossible with blood being blocked inside the penis by two different mechanisms (while inflow is not impeded) to have something that will yield to the increasing pressure. If something didn't yield, the penis would pop like an over-inflated balloon. I think that would explain a sudden loss in tunica elasticity.

Your experiment may provide some further insight into hypoxic growth, only wanted to highlight that it would not be out of the realm of possibility for this to be a pressure thing. Look at the cases of fractured penises, that's usually caused by a ruptured tunica so its not impervious to pressure deformation. Not many ways to find out for sure the causes, could even be a combination of hypoxic conditions and excessive pressure.

Keep up with your experiment and share your findings! Don't be afraid to discontinue if something goes wrong, protect yourself first.
 
I was thinking on this and when I was doing this experiment, in my own way, I was keeping an erection for at the minimum 2 hours, I would try to hold it for 4. I do not think one hour would cause the mega-phallus that would occur due to priapism.
 
doublelongdaddy;727624 said:
I was thinking on this and when I was doing this experiment, in my own way, I was keeping an erection for at the minimum 2 hours, I would try to hold it for 4. I do not think one hour would cause the mega-phallus that would occur due to priapism.

I think this may be a bit of anecdotal support towards a combination of both. Prolonged hypoxic conditions (reduced blood outflow to maintain erection but with some oxgen inflow) and extended episode of pressure (exerted from maintaining an erection).
 
All good points here. The thing about the penis reduction surgery megalophallus case that gets me is that it said the growth resulted after 3 separate priapisms. That's what led me to believe that it was a result of tissue growth. I've had ROCK hard PGE 1 erections before and no size gain immediately as in "tunica giving up". I really don't see how someone could get enough pressure to stretch the tunica just via pressure. I wish I did know tho, I'd do it tonite LOL
 
Xxxi;727665 said:
All good points here. The thing about the penis reduction surgery megalophallus case that gets me is that it said the growth resulted after 3 separate priapisms. That's what led me to believe that it was a result of tissue growth. I've had ROCK hard PGE 1 erections before and no size gain immediately as in "tunica giving up". I really don't see how someone could get enough pressure to stretch the tunica just via pressure. I wish I did know tho, I'd do it tonite LOL

Well see the main difference between your induced erection and priapism is that your "plumbing" still works. Your body won't allow damage to be done by pressure because your defense mechanisms are still functional. With priapism, something is mechanically blocking the path (sickled RBCs in the case of sickle cell anemia).

I'm gonna try to explain that concept in a way that makes it simpler for us. Let's say you have a water hose, you kink the hose and it slows down weather out flow and the pressure inside the tube is increased. This is a normal erection (doesn't matter if it's chemical or natural). If the pressure gets higher and higher the kink will loosen a bit to let more water out because it is the weak point. Now let's take something and block that hose internally. Let's take a ball (representing the clump sickled cells can form) that can stretch the malleable part of the hose but can't get past the thread connection (equivalent to venous valves in this example). No matter how much the pressure increases behind the ball, it won't let more water out than possible because it's clogged at the threaded connection (the strong point of the hose). It leaves only the weaker components of the hose to relieve that pressure. This would be like priapism. Did that make sense or did I fudge it up even more? Lol
 
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It is possible (in reference to the main post) but it is dangerous and can negatively affect and injure the penis if done improperly. Best we can do is clamping.
 
chargains;727668 said:
Well see the main difference between your induced erection and priapism is that your "plumbing" still works. Your body won't allow damage to be done by pressure because your defense mechanisms are still functional. With priapism, something is mechanically blocking the path (sickled RBCs in the case of sickle cell anemia).

I'm gonna try to explain that concept in a way that makes it simpler for us. Let's say you have a water hose, you kink the hose and it slows down weather out flow and the pressure inside the tube is increased. This is a normal erection (doesn't matter if it's chemical or natural). If the pressure gets higher and higher the kink will loosen a bit to let more water out because it is the weak point. Now let's take something and block that hose internally. Let's take a ball (representing the clump sickled cells can form) that can stretch the malleable part of the hose but can't get past the thread connection (equivalent to venous valves in this example). No matter how much the pressure increases behind the ball, it won't let more water out than possible because it's clogged at the threaded connection (the strong point of the hose). It leaves only the weaker components of the hose to relieve that pressure. This would be like priapism. Did that make sense or did I fudge it up even more? Lol


I do understand what you are saying. But back to the hose example, even if you totally kink it or put a stopper in it, it's not going to build up enough pressure to stretch the hose rubber (tunica). It's just going to be stagnant at the hoses expansion limit because the hose rubber is too stiff to expand just due to the pressure of the water. You'd need more water pressure to expand the hose beyond its pre-made diameter.

I think that is how the penis is. Even if you block off the outflow completely, I really don't see how you could get enough IN-FLOW PRESSURE to keep forcing more blood in. The tunica is soo tough and rigid, to have enough pressure to stretch it would have to be incredible pressure and I don't think the arteries are capable of pushing blood with that much force. I think the tunica has to be weakened by some other means first before mere blood pressure can expand it beyond its genetic limit.

I hope that made sense.

I mean, even with [words=http://www.mattersofsize.com/forum/penis-enlargement-forum/12539-slow-squash-jelq-nothing-give-me-better-expansion.html]ssj[/words], smashes, all kinds of intense clamping and compression, we still aren't able to force the tunica to "give up" and expand to massive sizes like some of these priapisms do. Do you think that the pressure from a heart pushing blood through an artery is really exerting more force than DLD when he's squashing his penis with the brute force of his hands?
 
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Xxxi;727684 said:
I do understand what you are saying. But back to the hose example, even if you totally kink it or put a stopper in it, it's not going to build up enough pressure to stretch the hose rubber (tunica). It's just going to be stagnant at the hoses expansion limit because the hose rubber is too stiff to expand just due to the pressure of the water. You'd need more water pressure to expand the hose beyond its pre-made diameter.

I think that is how the penis is. Even if you block off the outflow completely, I really don't see how you could get enough IN-FLOW PRESSURE to keep forcing more blood in. The tunica is soo tough and rigid, to have enough pressure to stretch it would have to be incredible pressure and I don't think the arteries are capable of pushing blood with that much force. I think the tunica has to be weakened by some other means first before mere blood pressure can expand it beyond its genetic limit.

I hope that made sense.

I mean, ever with [words=http://www.mattersofsize.com/forum/penis-enlargement-forum/12539-slow-squash-jelq-nothing-give-me-better-expansion.html]ssj[/words], smashes, all kinds of intense clamping and compression, we still aren't able to force the tunica to "give up" and expand to massive sizes like some of these priapisms do. Do you think that the pressure from a heart pushing blood through an artery is really exerting more force than DLD when he's squashing his penis with the brute force of his hands?

I agree. I only said that to illustrate the difference between a PGE-1 erection vs one caused by priapism. That wasn't speaking specifically about the deformation you're seeking.

Thats why I think it may be a combination effort that's causing the expansion. I don't think it is necessarily something to be attributed to one factor regardless of how significant each individual factor may seem. It's more like a cascade of events and conditions that have that outcome.

A little random information tho, the tensile strength of the average tunica is roughly 1500 mmHg. The heart's pressure in a normal person (not pressure measured peripherally in the arms or legs) is 10^4 Pascal ~ 750mmHg. Reaching tensile strength would rupture the tunica but the heart itself could get enough pressure for deformation if it wasn't impeded. Loss of elasticity would mean a sudden stretch from that build up. I don't think the force from hand work is less than what the heart is capable of but one is on a macro level and the other a micro level so pressure difference would be very different. [Also a difference in this example would be between water and blood. Blood has formed components that exert pressure in a different way from the molecules of water. Its part of the reason high blood pressure is damaging to microstructures.]

But yes, I can understand how you don't think it's just pressure, neither do I. I'm only saying that pressure can be a compounding factor in addition to whatever else is causing the change. Hopefully you can figure that out with your experiment.
 
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Thanks for the feedback Chargains. I really appreciate all your input and support. I think me and you both have realized that this could be the end-all of PE if we can finally nail it. I know some people are very against this kind of risky experimenting, but the thing that drives me is how CLOSE I feel like we are. We know it's between pressure and hypoxia. Those are the keys. If we can just figure out the correct way to apply those two factors, PE will never be the same again.

Haha and then if we could link up with DLD to make a user-friendly pump/clamp device that could keep the penis engorged while cutting off the oxygen in the right way to replicate megalophallus, boom, were multi millionaires!;) lol
 
Xxxi;727710 said:
Thanks for the feedback Chargains. I really appreciate all your input and support. I think me and you both have realized that this could be the end-all of PE if we can finally nail it. I know some people are very against this kind of risky experimenting, but the thing that drives me is how CLOSE I feel like we are. We know it's between pressure and hypoxia. Those are the keys. If we can just figure out the correct way to apply those two factors, PE will never be the same again.

Haha and then if we could link up with DLD to make a user-friendly pump/clamp device that could keep the penis engorged while cutting off the oxygen in the right way to replicate megalophallus, boom, were multi millionaires!;) lol

I really want you to find some success with this. Guys have been trying for a while without real notable progress with this type of growth. Its going to take some work to isolate the causes and replicate the conditions with minimum damage. You definitely sound like you don't mind putting in the work and that is very admirable. I don't have as much time as I would like to help research this but when I do, I will post it here as usual. I think the more that's added to the knowledge pool regarding this, the better we'll be able to target what the change would be.

That's why your experiment should provide some valuable insight into the hypoxic aspect and the growth associated with that. If you're successful, even marginally so, we as community could have better insight as to were to go from there. Like I keep saying lol, keep it up and bring your ideas, one day we'll find out what makes this work.
 
I have a pdf copy of an older case study from 2000 that I see referenced frequently in PE forums; however, they only show the abstract or the images. Does anyone know how I can upload the pdf to this thread?

Delete this link if it is against forum rules but this is a link that takes you to a direct download of the document. If you don't have an account with researchgate it will say something like "validating your browser" but it will still download the file as a pdf. https://www.researchgate.net/profil...evel-dependent-magnetic-resonance-imaging.pdf
 
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chargains;727655 said:
I think this may be a bit of anecdotal support towards a combination of both. Prolonged hypoxic conditions (reduced blood outflow to maintain erection but with some oxgen inflow) and extended episode of pressure (exerted from maintaining an erection).

You might be right. I spent a lot of time but did not produce.
 
For the guys who have already tried something similar, it may be helpful if you link your threads when you have time. I think it would be helpful to see what has already been tried.
 
I read that case that you posted. The strangest thing to me is that several cases I've read, the penis grew AFTER the priapism and over time to become huge. In this case however, it seems that the patient's penis was permanently enlarged DURING the priapism. That basically rules out the possibility of new tissue growth being the cause of his enlargement. I would love to be able to ask the guy how long the priapism lasted, and how hard the CC actually got before the tunica "gave up". Very interesting stuff. Basically shows us that if enough force is applied for the correct duration of time, you could literally gain inches of girth in a matter of a day. That's crazy imo.
 
The human body is wondrous to say the least. I really want to know more about the cases where the penis grows following the episode of priapism and growing larger over time, like some type of a repair and damage prevention mechanism. Hopefully I'll be able to learn more on that side of things thru this thread and some other research.
 
Xxxi;727684 said:
I do understand what you are saying. But back to the hose example, even if you totally kink it or put a stopper in it, it's not going to build up enough pressure to stretch the hose rubber (tunica). It's just going to be stagnant at the hoses expansion limit because the hose rubber is too stiff to expand just due to the pressure of the water. You'd need more water pressure to expand the hose beyond its pre-made diameter.

I think that is how the penis is. Even if you block off the outflow completely, I really don't see how you could get enough IN-FLOW PRESSURE to keep forcing more blood in. The tunica is soo tough and rigid, to have enough pressure to stretch it would have to be incredible pressure and I don't think the arteries are capable of pushing blood with that much force. I think the tunica has to be weakened by some other means first before mere blood pressure can expand it beyond its genetic limit.

I hope that made sense.

I mean, even with [words=http://www.mattersofsize.com/forum/penis-enlargement-forum/12539-slow-squash-jelq-nothing-give-me-better-expansion.html]ssj[/words], smashes, all kinds of intense clamping and compression, we still aren't able to force the tunica to "give up" and expand to massive sizes like some of these priapisms do. Do you think that the pressure from a heart pushing blood through an artery is really exerting more force than DLD when he's squashing his penis with the brute force of his hands?

The Hose will Expand after time.
The time is different for any Material. Steel, plastic or meshed Collagen.(Tunica)
The force applied over time is different for each but Every Material will give away at some Point.

Prolonged erection with more inflow then outflow(priapism) pressuring the Tunica will Expand the tunica if it goes beyond the elastic range.

If Low oxygen plays a part, maybe... Is it neccesary? No!
 
dickerschwanz;727940 said:
The Hose will Expand after time.
The time is different for any Material. Steel, plastic or meshed Collagen.(Tunica)
The force applied over time is different for each but Every Material will give away at some Point.

Prolonged erection with more inflow then outflow(priapism) pressuring the Tunica will Expand the tunica if it goes beyond the elastic range.

If Low oxygen plays a part, maybe... Is it neccesary? No!

This reminds me, I think its important to note what the tunica albuginea of the penis is composed of. The tunica is approximately 5% elastin with the remaining tissue being mostly collagen. It makes me think that there could even be an enzymatic reaction that causes a sudden loss of elasticity under pressure.
 
Exactly. It has to be some kind of chemical change in the cells by low oxygen that weakens them. Maybe they can't retain the energy they need to kee structure after a certain period of low oxygen. I still don't buy the idea that blood pressure in the CC gets high enough to do it alone. If that were the case then every priapism would result in megalophallus...but most actually do not.
 
chargains;727966 said:
This reminds me, I think its important to note what the tunica albuginea of the penis is composed of. The tunica is approximately 5% elastin with the remaining tissue being mostly collagen. It makes me think that there could even be an enzymatic reaction that causes a sudden loss of elasticity under pressure.

With such high density stretching becomes much more difficult but when it is stretched there is little chance of it ever returning to it's smaller size. Once stretched it is permanent.
 
Xxxi;727991 said:
Exactly. It has to be some kind of chemical change in the cells by low oxygen that weakens them. Maybe they can't retain the energy they need to kee structure after a certain period of low oxygen. I still don't buy the idea that blood pressure in the CC gets high enough to do it alone. If that were the case then every priapism would result in megalophallus...but most actually do not.
Many of the bouts of priapism causing these results last for days, not hours, and some have reoccurring episodes. This is why the time under tension principal works with [words=https://shop.mattersofsize.com/products/sizegenetics-penis-extender]extenders[/words], and also why [words=http://www.mattersofsize.com/forum/showthread.php?64036-My-Best-Work-Ever-Penis-Enlargement-will-Be-Changed-Forever-gt-gt-Gain%20inches%20with%20SRT-Theory-and-Routine]SRT[/words] is so effective. The tunica is tough, but is susceptible to fatigue like any living tissue, it seems to be a matter of finding that point of fatigue failure. Just be very careful because most cases of megalophallus result in a unusable dick, so ones that still function are pretty rare. Just my $0.02
 
There is elastic and plastic deformation with the tunica.
We want plastic deformation. Elastic means you return to initial size(like a rubber band).
Plastic you reach through a mix of time and intensity.
Between Low intensity Long time and short time and high intensity.
Heat influences it too.

Chemical actions Happen like water moving Out of the Collagen structure.

But as many gainers have shown there is no definite need to starve the tissue of oxygen.
The constant pressure of a priapism is putting the tunica under a Long time strain that at some point makes it reach the plastic stage.

You can gain by doing multiple Sets of clamping for some minutes intensly or by edging with Low intensity for an hour in the clamp. Maybe oxygen depletion happens under auch circumstances but it isn't the Prime Factor for the gains

I dont See how just starving tissue from oxygen alone helps.
You need some sort of expansion that reaches the plastic deformation stage.


IMO oxygen depletion in tissue is more an indicators of tissue death then gain.
Your time would be better Spend creating Quality expansion.
 
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ThatDude512;728137 said:
Many of the bouts of priapism causing these results last for days, not hours, and some have reoccurring episodes. This is why the time under tension principal works with [words=https://shop.mattersofsize.com/products/sizegenetics-penis-extender]extenders[/words], and also why [words=http://www.mattersofsize.com/forum/showthread.php?64036-My-Best-Work-Ever-Penis-Enlargement-will-Be-Changed-Forever-gt-gt-Gain%20inches%20with%20SRT-Theory-and-Routine]SRT[/words] is so effective. The tunica is tough, but is susceptible to fatigue like any living tissue, it seems to be a matter of finding that point of fatigue failure. Just be very careful because most cases of megalophallus result in a unusable dick, so ones that still function are pretty rare. Just my $0.02

I count this as 10¢ :) Great response!
 
dickerschwanz;728140 said:
There is elastic and plastic deformation with the tunica.
We want plastic deformation. Elastic means you return to initial size(like a rubber band).
Plastic you reach through a mix of time and intensity.
Between Low intensity Long time and short time and high intensity.
Heat influences it too.

Chemical actions Happen like water moving Out of the Collagen structure.

But as many gainers have shown there is no definite need to starve the tissue of oxygen.
The constant pressure of a priapism is putting the tunica under a Long time strain that at some point makes it reach the plastic stage.

You can gain by doing multiple Sets of clamping for some minutes intensly or by edging with Low intensity for an hour in the clamp. Maybe oxygen depletion happens under auch circumstances but it isn't the Prime Factor for the gains

I dont See how just starving tissue from oxygen alone helps.
You need some sort of expansion that reaches the plastic deformation stage.


IMO oxygen depletion in tissue is more an indicators of tissue death then gain.
Your time would be better Spend creating Quality expansion.

Very wise.
 
I think when it comes to priapism like conditions and replicating them gunning for a prolonged erection that is larger then normal is the first step.
It seems 4 hours is where one would come into priapism territory.
I think that's also the reason why pharmaceutical companies hint at "Call your doctor" if you have an erection lasting longer then 4 hours.

If I would do that I would look to get more and more time added day by day. If you do such endeavour cold start bad tissue might be the end result.
 
I agree with much of what's been said recently. I have changed my approach and getting results already. I don't have a ton of time rn but I will explain later what I'm doing now. Thanks to everyone for the input! You guys are incredible.
 
dickerschwanz;728258 said:
I think when it comes to priapism like conditions and replicating them gunning for a prolonged erection that is larger then normal is the first step.
It seems 4 hours is where one would come into priapism territory.
I think that's also the reason why pharmaceutical companies hint at "Call your doctor" if you have an erection lasting longer then 4 hours.

If I would do that I would look to get more and more time added day by day. If you do such endeavour cold start bad tissue might be the end result.

I could never make it that long every day, simply too much for me, I tried and tried but I could not go beyond the 4 hours.
 
Xxxi;728274 said:
I agree with much of what's been said recently. I have changed my approach and getting results already. I don't have a ton of time rn but I will explain later what I'm doing now. Thanks to everyone for the input! You guys are incredible.

Can't wait to hear what you have found out, should be interesting!
 
Hey guys, been super busy lately. Broke up with my gf and moved out of where I was staying. So I've been pretty far off my routine lately. The good news is that the chick I've been sleeping with this week has commented on my size several times and was even scared of it the first time. Never been told that before,, so something is working. Just trying to find the silver lining in the whole break up situation and let this chick keep my mind off the one that hurt me. But whatever, wanted to let ya know why I haven't replied much.

Anyway, all this talk about the tunica limit interested me in finding that stress limit and trying to distort the tunica through stress. What I've been doing is getting totally hard, then squeeze the blood only out of the glans and then clamp CRAZY TIGHT at the base with a quick release cable clamp. I do this while sitting on the toilet (seat down of course.). Then I push my dick flat against the seat and press down on the mid/upper shaft with both palms placed one on top of the other as if I'm doing CPR. However, I'm pressing down as hard as possible. Basically using all my body weight almost (182lbs). This forces the CC literally ROCK HARD. Kinda scary actually. And I'll hold that press for 15-20 sec. I'll do that as many times as I feel necessary. The cool part is, from what I've seen, gains this way are permanent. My penis remains slightly wider after the session and I've noticed how much wider it's beginning to look during sex too. Im gonna keep this up and report back to you guys. I'm still gonna play with oxygen manipulation too when I have time. But for now this INCREDIBLY intense pressure is working!! Give it a try if your brace enough. The key is having the clamp soo tight that u can press crazy hard without any blood escaping. You should feel the CC hard as nails and it should look wider after a few 20 sec presses. Let me know if any of you all try this!
 
Xxxi;728423 said:
Hey guys, been super busy lately. Broke up with my gf and moved out of where I was staying. So I've been pretty far off my routine lately. The good news is that the chick I've been sleeping with this week has commented on my size several times and was even scared of it the first time. Never been told that before,, so something is working. Just trying to find the silver lining in the whole break up situation and let this chick keep my mind off the one that hurt me. But whatever, wanted to let ya know why I haven't replied much.

Anyway, all this talk about the tunica limit interested me in finding that stress limit and trying to distort the tunica through stress. What I've been doing is getting totally hard, then squeeze the blood only out of the glans and then clamp CRAZY TIGHT at the base with a quick release cable clamp. I do this while sitting on the toilet (seat down of course.). Then I push my dick flat against the seat and press down on the mid/upper shaft with both palms placed one on top of the other as if I'm doing CPR. However, I'm pressing down as hard as possible. Basically using all my body weight almost (182lbs). This forces the CC literally ROCK HARD. Kinda scary actually. And I'll hold that press for 15-20 sec. I'll do that as many times as I feel necessary. The cool part is, from what I've seen, gains this way are permanent. My penis remains slightly wider after the session and I've noticed how much wider it's beginning to look during sex too. Im gonna keep this up and report back to you guys. I'm still gonna play with oxygen manipulation too when I have time. But for now this INCREDIBLY intense pressure is working!! Give it a try if your brace enough. The key is having the clamp soo tight that u can press crazy hard without any blood escaping. You should feel the CC hard as nails and it should look wider after a few 20 sec presses. Let me know if any of you all try this!

Sounds interesting the only thing that would worry me is Thrombosis and Discoloration. That would be quite a bit of pressure so it would be very important to keep a close eye on things so issues do not arise. I like that you have the guts to go through this and I think many are rooting for you to come with some good results. Just be careful!
 
Xxxi;726255 said:
Hey guys, I don't know if this exact way of thinking has been discussed lately but it seems that people aren't doing it so I'd like to bring it to light.

It seems that when we talk about girth gains, we tend to talk about stretching the tunica. However, Im starting to think there might be a better way. Many of you are probably familiar with the medical literature on the first "penis reduction surgery". If you aren't familiar with it, to sum it up for ya: a 17 year old suffered a couple episodes of priapism and developed a functional megalophallus nearly the size of a football. Sex was impossible for him because of the size, so he got a penis reduction surgery. The penis was fully functional though despite its size. Just too big for penetration into a vagina.

NOW, the interesting part to me is this.. the doctors of that surgery said in the article that the penis had massive amounts of HEALTHY, FIBROUS BLEEDING tissue and smooth muscle. It was not just an overly stretched tunica that could hold more blood. It was actually new tissue growth that resulted from hypoxia (reduced blood oxygen). It's also important to note that his penis grew to this insane size FOLLOWING the priapism, not during.

We know that hypoxia results in growth of collagen and smooth muscle. So why are we not more focused on cutting off blood oxygen more often (for short periods) with the goal of stimulating hypoxic growth of new tissue?

Maybe something along the lines of tying off an erection until the penis turns purple and then releasing for a few minutes and repeating this several times per session to stimulate tissue growth? Any thoughts on this??

Thanks!

This is so funny. DO not look for shortcut to gains bro. Don't try this at all.
 
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