Yes testosterone will make a woman's clit bigger but that's completely different than when a man takes it. Because women have extremely low amounts of natural test, injecting it into their body forces changes to occur. Comparing a man taking testosterone injections and women taking testosterone injections is apples to oranges...sasquatch said:
I think you mean male-to-female transgenders... transvestites are basically cross-dressers. A male trying to change into a female would be taking female hormones (estrogen), not testosterone.And how do the male to female transvestites get those insanely huge dicks (some, not all) with all the hormonal manipulation ?
Totally agree... i've been on cycles where i was injecting over 1 gram of test per week while doing manual Penis Enlargement and didn't notice faster gains than when i was off-cycle.AlbertaBeef said:There is no way I can see test making your dick bigger. You may get hornier and get better erections but there is no way it can increase your size. If such was the case these juice monsters taking 2000 grams of test a week would be hung like young bull elephants and judging by the way ol' Ronny fill out his trunks in the Mr Olympia I don't think it happens
fallguy said:
Bro, you're right in the age zone where most men experience a huge decline in endogenous testosterone levels. You should go to a doctor and have your test levels checked... if it's too low, your doctor will prescribe Hormone Replacement Therapy (HRT) or more specifically Testosterone Replacement Therapy (TRT) to get you bacl to normal levels.fallguy said:
sikdogg said:
550 is not bad at all... but without knowing the lab range for you test it's hard to say for sure. Most lab ranges run 350-1000 for Total Testosterone. Your result falls just below the mid so it's good. I don't think any doctor would treat you for low test with that result. Most doctors that treat hypogonadal men (men with low test) aim for getting Total Test levels in the upper 1/3 of the range or somewhere above 700. At this level, you will feel like you're 25 years old again, physically and libido will definitely be up.kcee said:
yes and tsh, t3, t4, and prolactin are also importantsikdogg said:
I think test's penile growth effects wear off after puberty because of androgen receptor down-regulation, after a few years of high exposure. Theoretically, if you can up-regulate androgen receptors in the penis, you can spur growth with test (if this theory is true). Prostaglandin E-1 is one substance known to upregulate androgen receptors. Bodybuilders who locally inject pge1 notice the area grow much faster than other body parts, and it makes the following steroid cycle very effective when working that specific area even after discontinuing the pge1.sikdogg said:
That theory has actually been de-bunked... If an adolescent's AR down-regulates it does so at the same time as that the epiphyseal growth-plates close. An adult male's AR does not down-regulate after years of high exposure. This is proven everyday by professional bodybuilders who routinely take several grams of AAS per week for 10 years or more. If that was the case, no professional bodybuilder could continue to make gains year after year.goldmember said:
If you are experiencing symptoms of low testosterone, then your doctor should be putting you on TRT. Go to All Things Male and download Dr. Crislers paper TRT: A Recipe for Success. Read it and show it to your doctor. If he still won't treat you then you need to find a new doctor.fallguy said:
Receptors can down-regulate locally. It is possible that they down-regulate in the smooth muscle (CC) while up-regulating in the skeletal muscle.sikdogg said:
I'm not necessarily saying that they do but speculate that the closure of the epiphyseal plates is part of a larger physiology change that takes place which effect how hormones affect the body. It is sort of a demarcation point from which testosterone no longer exerts its effects on genitals and GH no longer exerts its effect on growth. I don't pretent to fully understand the whole metabolic process that happens when one passes puberty, just guesses from past study. I could be way off base...goldmember said:
This study seems to be suggesting that DHT upregulates the conversion of test-->estro....sikdogg said:
Actually i think it says that the test-to-estro conversion was blocked by the inhibition of test-to-DHT conversion by using a 5-AR inhibitor... now i'm a little lost. Maybe your theory was right...goldmember said:
Gotcha... i assumed when you mentioned "the most androgenic steroid..." you meant DHT. My bad...
Arimidex plus finasteride should raise test levels while shrinking the prostate. The biggest cause of prostate enlargement is estrogen in the presence of 5-LO enzyme. Arimidex will lower estrogen (very significantly) and finasteride will lower DHT (but not test). Sikdogg pointed out a study that shows finasteride along with arimidex may lower estrogen levels even better than arimidex alone...goinfor11x7 said:Is there any way to raise your Test. levels naturally without taking Test. supplements? My urologist has me on a med. to shrink my prostate which is two and half times the normal size. So I hardly need worry about raising my Test. levels when the doc. wants to lower them. I hope Kcee thinks about this before trying to raise his Test. levels being that he's into his 50's now.
I asked the doc. if the med. would shrink my penis as well. He just stared at me. Not very enlightened about discussing these matters. Oh, how I wish I had my old urologist back!!!
<
This must be a popular guide to TRT because I remember reading this about two years ago, before I even belonged to this forum. A very good read, and any middle aged man should read it, regardless of whether or not they are experiencing symptoms of low testosterone.sikdogg said:
No...this isn't MY theory. It actually seems to be pretty well-accepted among steroid circles that the androgenicity of the steroid determines the severity of the side effects and the level of aromatization. The more androgenic the steroid, the more it causes bloating, acne, liver damage, hair loss, virilization, gyno, HPTA down-regulation, and (a positive) strength gains. The more anabolic the steroid, the less you notice the aforementioned side effects and the better the protein sysnthesis abilities. Ironically, HPTA down-regulation is effected very little by testosterone levels themself, but rather by ESTROGEN. Highly androgenic steroids convert readily to estrogen, and therein lies the main reason for HPTA down-regulation caused by androgenic steroids. Another paradox is that in order for an anabolic steroid to have a significant benefit, there must be at least a small level of circulating estrogen in the blood...this explains why testosterones are more effective for mass and strength cycles than DHT (which is usually better as a hardening agent).sikdogg said:
I think DHT is 16 times more androgenic than it is anabolic (1:16), and you are right in saying it is the most androgenic steroid. However, DHT is not able to aromatize, which explains its use as a hardening agent. That confusion was my fault, but look at how much we have discovered because I screwed up and you go throwing out that study on 5a-reductase/AI...lolsikdogg said:
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